| pubmed-article:20864514 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:20864514 | lifeskim:mentions | umls-concept:C0021368 | lld:lifeskim |
| pubmed-article:20864514 | lifeskim:mentions | umls-concept:C0026809 | lld:lifeskim |
| pubmed-article:20864514 | lifeskim:mentions | umls-concept:C2711227 | lld:lifeskim |
| pubmed-article:20864514 | lifeskim:mentions | umls-concept:C1335073 | lld:lifeskim |
| pubmed-article:20864514 | lifeskim:mentions | umls-concept:C0021655 | lld:lifeskim |
| pubmed-article:20864514 | lifeskim:mentions | umls-concept:C1417830 | lld:lifeskim |
| pubmed-article:20864514 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:20864514 | pubmed:dateCreated | 2011-1-3 | lld:pubmed |
| pubmed-article:20864514 | pubmed:abstractText | The nuclear receptor TAK1/TR4/NR2C2 is expressed in several tissues that are important in the control of energy homeostasis. In this study, we investigate whether TAK1 functions as a regulator of lipid and energy homeostasis and has a role in metabolic syndrome. | lld:pubmed |
| pubmed-article:20864514 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20864514 | pubmed:language | eng | lld:pubmed |
| pubmed-article:20864514 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20864514 | pubmed:citationSubset | AIM | lld:pubmed |
| pubmed-article:20864514 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20864514 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20864514 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20864514 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20864514 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20864514 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20864514 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20864514 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:20864514 | pubmed:month | Jan | lld:pubmed |
| pubmed-article:20864514 | pubmed:issn | 1939-327X | lld:pubmed |
| pubmed-article:20864514 | pubmed:author | pubmed-author:GaoH YHY | lld:pubmed |
| pubmed-article:20864514 | pubmed:author | pubmed-author:KimYong-SikYS | lld:pubmed |
| pubmed-article:20864514 | pubmed:author | pubmed-author:JettenAnton... | lld:pubmed |
| pubmed-article:20864514 | pubmed:author | pubmed-author:BortnerCarl... | lld:pubmed |
| pubmed-article:20864514 | pubmed:author | pubmed-author:YangXiao-Ping... | lld:pubmed |
| pubmed-article:20864514 | pubmed:author | pubmed-author:KangHong... | lld:pubmed |
| pubmed-article:20864514 | pubmed:author | pubmed-author:OkamotoKyokoK | lld:pubmed |
| pubmed-article:20864514 | pubmed:author | pubmed-author:TakedaYukimas... | lld:pubmed |
| pubmed-article:20864514 | pubmed:author | pubmed-author:LiaoGraceG | lld:pubmed |
| pubmed-article:20864514 | pubmed:author | pubmed-author:WadaTairaT | lld:pubmed |
| pubmed-article:20864514 | pubmed:author | pubmed-author:DangHuaixinH | lld:pubmed |
| pubmed-article:20864514 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:20864514 | pubmed:volume | 60 | lld:pubmed |
| pubmed-article:20864514 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:20864514 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:20864514 | pubmed:pagination | 177-88 | lld:pubmed |
| pubmed-article:20864514 | pubmed:dateRevised | 2011-10-3 | lld:pubmed |
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| pubmed-article:20864514 | pubmed:meshHeading | pubmed-meshheading:20864514... | lld:pubmed |
| pubmed-article:20864514 | pubmed:meshHeading | pubmed-meshheading:20864514... | lld:pubmed |
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| pubmed-article:20864514 | pubmed:meshHeading | pubmed-meshheading:20864514... | lld:pubmed |
| pubmed-article:20864514 | pubmed:meshHeading | pubmed-meshheading:20864514... | lld:pubmed |
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| pubmed-article:20864514 | pubmed:meshHeading | pubmed-meshheading:20864514... | lld:pubmed |
| pubmed-article:20864514 | pubmed:year | 2011 | lld:pubmed |
| pubmed-article:20864514 | pubmed:articleTitle | Nuclear orphan receptor TAK1/TR4-deficient mice are protected against obesity-linked inflammation, hepatic steatosis, and insulin resistance. | lld:pubmed |
| pubmed-article:20864514 | pubmed:affiliation | Cell Biology Section, Laboratory of Respiratory Biology, National Institute of Environmental Health Sciences, National Institutes of Health, Research Triangle Park, North Carolina, USA. | lld:pubmed |
| pubmed-article:20864514 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:20864514 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
| pubmed-article:20864514 | pubmed:publicationType | Research Support, N.I.H., Intramural | lld:pubmed |
| entrez-gene:22026 | entrezgene:pubmed | pubmed-article:20864514 | lld:entrezgene |
| http://linkedlifedata.com/r... | entrezgene:pubmed | pubmed-article:20864514 | lld:entrezgene |
