Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
1
pubmed:dateCreated
2011-1-3
pubmed:abstractText
The nuclear receptor TAK1/TR4/NR2C2 is expressed in several tissues that are important in the control of energy homeostasis. In this study, we investigate whether TAK1 functions as a regulator of lipid and energy homeostasis and has a role in metabolic syndrome.
pubmed:grant
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
AIM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Jan
pubmed:issn
1939-327X
pubmed:author
pubmed:issnType
Electronic
pubmed:volume
60
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
177-88
pubmed:dateRevised
2011-10-3
pubmed:meshHeading
pubmed-meshheading:20864514-Adaptor Proteins, Signal Transducing, pubmed-meshheading:20864514-Adipose Tissue, pubmed-meshheading:20864514-Animals, pubmed-meshheading:20864514-Dietary Fats, pubmed-meshheading:20864514-Epididymis, pubmed-meshheading:20864514-Fatty Liver, pubmed-meshheading:20864514-Flow Cytometry, pubmed-meshheading:20864514-Inflammation, pubmed-meshheading:20864514-Insulin Resistance, pubmed-meshheading:20864514-Male, pubmed-meshheading:20864514-Metabolic Syndrome X, pubmed-meshheading:20864514-Mice, pubmed-meshheading:20864514-Mice, Knockout, pubmed-meshheading:20864514-Obesity, pubmed-meshheading:20864514-Organ Size, pubmed-meshheading:20864514-RNA, pubmed-meshheading:20864514-Receptors, Steroid, pubmed-meshheading:20864514-Receptors, Thyroid Hormone, pubmed-meshheading:20864514-Reverse Transcriptase Polymerase Chain Reaction
pubmed:year
2011
pubmed:articleTitle
Nuclear orphan receptor TAK1/TR4-deficient mice are protected against obesity-linked inflammation, hepatic steatosis, and insulin resistance.
pubmed:affiliation
Cell Biology Section, Laboratory of Respiratory Biology, National Institute of Environmental Health Sciences, National Institutes of Health, Research Triangle Park, North Carolina, USA.
pubmed:publicationType
Journal Article, Research Support, N.I.H., Extramural, Research Support, N.I.H., Intramural