pubmed-article:20861649 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:20861649 | lifeskim:mentions | umls-concept:C0002390 | lld:lifeskim |
pubmed-article:20861649 | lifeskim:mentions | umls-concept:C0026336 | lld:lifeskim |
pubmed-article:20861649 | lifeskim:mentions | umls-concept:C0301872 | lld:lifeskim |
pubmed-article:20861649 | lifeskim:mentions | umls-concept:C0205191 | lld:lifeskim |
pubmed-article:20861649 | lifeskim:mentions | umls-concept:C0591833 | lld:lifeskim |
pubmed-article:20861649 | pubmed:issue | 3 | lld:pubmed |
pubmed-article:20861649 | pubmed:dateCreated | 2011-2-21 | lld:pubmed |
pubmed-article:20861649 | pubmed:abstractText | Chronic hypersensitivity pneumonitis (HP) can lead to irreversible pulmonary fibrosis. A good animal model is essential to elucidate the mechanisms of this disease. We previously reported that a Th2 predominance may play an important role in the fibrogenesis in chronic HP patients. A study was undertaken to evaluate whether Th2-biased immune responses were crucial during the processes of lung fibrosis in a murine model of chronic HP. | lld:pubmed |
pubmed-article:20861649 | pubmed:language | eng | lld:pubmed |
pubmed-article:20861649 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20861649 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:20861649 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20861649 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:20861649 | pubmed:issn | 1423-0097 | lld:pubmed |
pubmed-article:20861649 | pubmed:author | pubmed-author:MiyakeShujiS | lld:pubmed |
pubmed-article:20861649 | pubmed:author | pubmed-author:InaseNaohikoN | lld:pubmed |
pubmed-article:20861649 | pubmed:author | pubmed-author:YoshizawaYasu... | lld:pubmed |
pubmed-article:20861649 | pubmed:author | pubmed-author:MiyazakiYasun... | lld:pubmed |
pubmed-article:20861649 | pubmed:author | pubmed-author:FuruieMasashi... | lld:pubmed |
pubmed-article:20861649 | pubmed:author | pubmed-author:YasuiMakitoM | lld:pubmed |
pubmed-article:20861649 | pubmed:author | pubmed-author:MitakaKeikoK | lld:pubmed |
pubmed-article:20861649 | pubmed:copyrightInfo | Copyright © 2010 S. Karger AG, Basel. | lld:pubmed |
pubmed-article:20861649 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:20861649 | pubmed:volume | 154 | lld:pubmed |
pubmed-article:20861649 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:20861649 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:20861649 | pubmed:pagination | 264-74 | lld:pubmed |
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pubmed-article:20861649 | pubmed:year | 2011 | lld:pubmed |
pubmed-article:20861649 | pubmed:articleTitle | Th2-biased immune responses are important in a murine model of chronic hypersensitivity pneumonitis. | lld:pubmed |
pubmed-article:20861649 | pubmed:affiliation | Department of Integrated Pulmonology, Tokyo Medical and Dental University, Tokyo, Japan. | lld:pubmed |
pubmed-article:20861649 | pubmed:publicationType | Journal Article | lld:pubmed |
entrez-gene:116553 | entrezgene:pubmed | pubmed-article:20861649 | lld:entrezgene |
entrez-gene:287287 | entrezgene:pubmed | pubmed-article:20861649 | lld:entrezgene |