Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
2
pubmed:dateCreated
2011-1-13
pubmed:abstractText
Ginsenoside-Rg1, the most prevalent active constituent of Panax ginseng, has been shown to possess potent pro-angiogenic properties and therefore poses special interest for the development as a novel modality for angiotherapy. Rg1 can activate the glucocorticoid receptor (GR). However, the mechanism that transmits these pro-angiogenic effects is still unclear.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
http://linkedlifedata.com/resource/pubmed/chemical/Angiogenesis Inducing Agents, http://linkedlifedata.com/resource/pubmed/chemical/FGFR1 protein, human, http://linkedlifedata.com/resource/pubmed/chemical/Ginsenosides, http://linkedlifedata.com/resource/pubmed/chemical/Hormone Antagonists, http://linkedlifedata.com/resource/pubmed/chemical/Mifepristone, http://linkedlifedata.com/resource/pubmed/chemical/NOS3 protein, human, http://linkedlifedata.com/resource/pubmed/chemical/NR3C1 protein, human, http://linkedlifedata.com/resource/pubmed/chemical/Nitric Oxide Synthase Type III, http://linkedlifedata.com/resource/pubmed/chemical/Phosphatidylinositol 3-Kinase, http://linkedlifedata.com/resource/pubmed/chemical/Protein Kinase Inhibitors, http://linkedlifedata.com/resource/pubmed/chemical/Proto-Oncogene Proteins c-akt, http://linkedlifedata.com/resource/pubmed/chemical/Pyrroles, http://linkedlifedata.com/resource/pubmed/chemical/Receptor, Fibroblast Growth..., http://linkedlifedata.com/resource/pubmed/chemical/Receptors, Glucocorticoid, http://linkedlifedata.com/resource/pubmed/chemical/SU 5402, http://linkedlifedata.com/resource/pubmed/chemical/ginsenoside Rg1
pubmed:status
MEDLINE
pubmed:month
Feb
pubmed:issn
1755-3245
pubmed:author
pubmed:issnType
Electronic
pubmed:day
1
pubmed:volume
89
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
419-25
pubmed:meshHeading
pubmed-meshheading:20855522-Angiogenesis Inducing Agents, pubmed-meshheading:20855522-Cells, Cultured, pubmed-meshheading:20855522-Endothelial Cells, pubmed-meshheading:20855522-Enzyme Activation, pubmed-meshheading:20855522-Ginsenosides, pubmed-meshheading:20855522-Hormone Antagonists, pubmed-meshheading:20855522-Humans, pubmed-meshheading:20855522-Mifepristone, pubmed-meshheading:20855522-Neovascularization, Physiologic, pubmed-meshheading:20855522-Nitric Oxide Synthase Type III, pubmed-meshheading:20855522-Phosphatidylinositol 3-Kinase, pubmed-meshheading:20855522-Phosphorylation, pubmed-meshheading:20855522-Protein Kinase Inhibitors, pubmed-meshheading:20855522-Proto-Oncogene Proteins c-akt, pubmed-meshheading:20855522-Pyrroles, pubmed-meshheading:20855522-RNA Interference, pubmed-meshheading:20855522-Receptor, Fibroblast Growth Factor, Type 1, pubmed-meshheading:20855522-Receptor Cross-Talk, pubmed-meshheading:20855522-Receptors, Glucocorticoid
pubmed:year
2011
pubmed:articleTitle
Ginsenoside-Rg1 induces angiogenesis via non-genomic crosstalk of glucocorticoid receptor and fibroblast growth factor receptor-1.
pubmed:affiliation
School of Biological Sciences, University of Hong Kong, 4S-14 Kadoorie Biological Sciences Building, Pokfulam Road, Hong Kong.
pubmed:publicationType
Journal Article, Research Support, Non-U.S. Gov't