| pubmed-article:20735837 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:20735837 | lifeskim:mentions | umls-concept:C0332307 | lld:lifeskim |
| pubmed-article:20735837 | lifeskim:mentions | umls-concept:C1280551 | lld:lifeskim |
| pubmed-article:20735837 | lifeskim:mentions | umls-concept:C0011854 | lld:lifeskim |
| pubmed-article:20735837 | lifeskim:mentions | umls-concept:C0232164 | lld:lifeskim |
| pubmed-article:20735837 | lifeskim:mentions | umls-concept:C0025519 | lld:lifeskim |
| pubmed-article:20735837 | lifeskim:mentions | umls-concept:C0017263 | lld:lifeskim |
| pubmed-article:20735837 | lifeskim:mentions | umls-concept:C0851827 | lld:lifeskim |
| pubmed-article:20735837 | lifeskim:mentions | umls-concept:C1701901 | lld:lifeskim |
| pubmed-article:20735837 | pubmed:dateCreated | 2010-9-10 | lld:pubmed |
| pubmed-article:20735837 | pubmed:abstractText | The mechanisms responsible for the cardiovascular mortality in type I diabetes (DM) have not been defined completely. We have shown in conscious dogs with DM that: 1) baseline coronary blood flow (CBF) was significantly decreased, 2) endothelium-dependent (ACh) coronary vasodilation was impaired, and 3) reflex cholinergic NO-dependent coronary vasodilation was selectively depressed. The most likely mechanism responsible for the depressed reflex cholinergic NO-dependent coronary vasodilation was the decreased bioactivity of NO from the vascular endothelium. The goal of this study was to investigate changes in cardiac gene expression in a canine model of alloxan-induced type 1 diabetes. | lld:pubmed |
| pubmed-article:20735837 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20735837 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20735837 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20735837 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20735837 | pubmed:language | eng | lld:pubmed |
| pubmed-article:20735837 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20735837 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:20735837 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20735837 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20735837 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:20735837 | pubmed:issn | 1475-2840 | lld:pubmed |
| pubmed-article:20735837 | pubmed:author | pubmed-author:RecchiaFabio... | lld:pubmed |
| pubmed-article:20735837 | pubmed:author | pubmed-author:HintzeThomas... | lld:pubmed |
| pubmed-article:20735837 | pubmed:author | pubmed-author:KinugawaShint... | lld:pubmed |
| pubmed-article:20735837 | pubmed:author | pubmed-author:OjaimiCarolin... | lld:pubmed |
| pubmed-article:20735837 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:20735837 | pubmed:volume | 9 | lld:pubmed |
| pubmed-article:20735837 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:20735837 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:20735837 | pubmed:pagination | 43 | lld:pubmed |
| pubmed-article:20735837 | pubmed:meshHeading | pubmed-meshheading:20735837... | lld:pubmed |
| pubmed-article:20735837 | pubmed:meshHeading | pubmed-meshheading:20735837... | lld:pubmed |
| pubmed-article:20735837 | pubmed:meshHeading | pubmed-meshheading:20735837... | lld:pubmed |
| pubmed-article:20735837 | pubmed:meshHeading | pubmed-meshheading:20735837... | lld:pubmed |
| pubmed-article:20735837 | pubmed:meshHeading | pubmed-meshheading:20735837... | lld:pubmed |
| pubmed-article:20735837 | pubmed:meshHeading | pubmed-meshheading:20735837... | lld:pubmed |
| pubmed-article:20735837 | pubmed:meshHeading | pubmed-meshheading:20735837... | lld:pubmed |
| pubmed-article:20735837 | pubmed:meshHeading | pubmed-meshheading:20735837... | lld:pubmed |
| pubmed-article:20735837 | pubmed:meshHeading | pubmed-meshheading:20735837... | lld:pubmed |
| pubmed-article:20735837 | pubmed:meshHeading | pubmed-meshheading:20735837... | lld:pubmed |
| pubmed-article:20735837 | pubmed:meshHeading | pubmed-meshheading:20735837... | lld:pubmed |
| pubmed-article:20735837 | pubmed:meshHeading | pubmed-meshheading:20735837... | lld:pubmed |
| pubmed-article:20735837 | pubmed:meshHeading | pubmed-meshheading:20735837... | lld:pubmed |
| pubmed-article:20735837 | pubmed:meshHeading | pubmed-meshheading:20735837... | lld:pubmed |
| pubmed-article:20735837 | pubmed:year | 2010 | lld:pubmed |
| pubmed-article:20735837 | pubmed:articleTitle | Oxidant-NO dependent gene regulation in dogs with type I diabetes: impact on cardiac function and metabolism. | lld:pubmed |
| pubmed-article:20735837 | pubmed:affiliation | Department of Physiology, New York Medical College, Valhalla, NY 10595, USA. caroline_ojaimi@nymc.edu | lld:pubmed |
| pubmed-article:20735837 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:20735837 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
