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pubmed-article:20713897pubmed:abstractTextDilated cardiomyopathy (DCM), characterized by dilatation and dysfunction of the left ventricle, is an important cause of heart failure. Many mutations in various genes, including cytoskeletal protein genes and contractile protein genes, have been identified in DCM patients, but the mechanisms of how such mutations lead to DCM remain unknown.lld:pubmed
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pubmed-article:20713897pubmed:articleTitleCa2+/calmodulin-dependent kinase IIdelta causes heart failure by accumulation of p53 in dilated cardiomyopathy.lld:pubmed
pubmed-article:20713897pubmed:affiliationDepartment of Cardiovascular Science and Medicine, Chiba University Graduate School of Medicine, Chiba, Japan.lld:pubmed
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