Linked Life Data

20713897

Source:http://linkedlifedata.com/resource/pubmed/id/20713897

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
9
pubmed:dateCreated
2010-8-31
pubmed:abstractText
Dilated cardiomyopathy (DCM), characterized by dilatation and dysfunction of the left ventricle, is an important cause of heart failure. Many mutations in various genes, including cytoskeletal protein genes and contractile protein genes, have been identified in DCM patients, but the mechanisms of how such mutations lead to DCM remain unknown.
pubmed:grant
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
AIM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Aug
pubmed:issn
1524-4539
pubmed:author
pubmed:issnType
Electronic
pubmed:day
31
pubmed:volume
122
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
891-9
pubmed:dateRevised
2011-11-17
pubmed:meshHeading
pubmed-meshheading:20713897-Actin Cytoskeleton, pubmed-meshheading:20713897-Actins, pubmed-meshheading:20713897-Animals, pubmed-meshheading:20713897-Apoptosis, pubmed-meshheading:20713897-Benzylamines, pubmed-meshheading:20713897-Calcium, pubmed-meshheading:20713897-Calcium-Calmodulin-Dependent Protein Kinase Type 2, pubmed-meshheading:20713897-Cardiomyopathy, Dilated, pubmed-meshheading:20713897-Disease Models, Animal, pubmed-meshheading:20713897-Enzyme Activation, pubmed-meshheading:20713897-Heart Failure, pubmed-meshheading:20713897-Humans, pubmed-meshheading:20713897-Mice, pubmed-meshheading:20713897-Mice, Transgenic, pubmed-meshheading:20713897-Myocytes, Cardiac, pubmed-meshheading:20713897-Protein Kinase Inhibitors, pubmed-meshheading:20713897-Sulfonamides, pubmed-meshheading:20713897-Tumor Suppressor Protein p53
pubmed:year
2010
pubmed:articleTitle
Ca2+/calmodulin-dependent kinase IIdelta causes heart failure by accumulation of p53 in dilated cardiomyopathy.
pubmed:affiliation
Department of Cardiovascular Science and Medicine, Chiba University Graduate School of Medicine, Chiba, Japan.
pubmed:publicationType
Journal Article, Research Support, Non-U.S. Gov't, Research Support, N.I.H., Extramural