pubmed-article:20682682 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:20682682 | lifeskim:mentions | umls-concept:C0927232 | lld:lifeskim |
pubmed-article:20682682 | lifeskim:mentions | umls-concept:C0205054 | lld:lifeskim |
pubmed-article:20682682 | lifeskim:mentions | umls-concept:C0040615 | lld:lifeskim |
pubmed-article:20682682 | lifeskim:mentions | umls-concept:C0021655 | lld:lifeskim |
pubmed-article:20682682 | lifeskim:mentions | umls-concept:C0011209 | lld:lifeskim |
pubmed-article:20682682 | lifeskim:mentions | umls-concept:C0171023 | lld:lifeskim |
pubmed-article:20682682 | lifeskim:mentions | umls-concept:C0205263 | lld:lifeskim |
pubmed-article:20682682 | pubmed:issue | 10 | lld:pubmed |
pubmed-article:20682682 | pubmed:dateCreated | 2010-9-29 | lld:pubmed |
pubmed-article:20682682 | pubmed:abstractText | Olanzapine (OLZ) is an atypical antipsychotic whose clinical efficacy is hampered by side effects including weight gain and diabetes. Recent evidence shows that OLZ alters insulin sensitivity independent of changes in body weight and composition. The present study addresses whether OLZ-induced insulin resistance is driven by its central actions. | lld:pubmed |
pubmed-article:20682682 | pubmed:language | eng | lld:pubmed |
pubmed-article:20682682 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20682682 | pubmed:citationSubset | AIM | lld:pubmed |
pubmed-article:20682682 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20682682 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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pubmed-article:20682682 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20682682 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20682682 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20682682 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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pubmed-article:20682682 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:20682682 | pubmed:month | Oct | lld:pubmed |
pubmed-article:20682682 | pubmed:issn | 1939-327X | lld:pubmed |
pubmed-article:20682682 | pubmed:author | pubmed-author:ObiciSilvanaS | lld:pubmed |
pubmed-article:20682682 | pubmed:author | pubmed-author:HaasMichaelM | lld:pubmed |
pubmed-article:20682682 | pubmed:author | pubmed-author:MartinsPaulo... | lld:pubmed |
pubmed-article:20682682 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:20682682 | pubmed:volume | 59 | lld:pubmed |
pubmed-article:20682682 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:20682682 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:20682682 | pubmed:pagination | 2418-25 | lld:pubmed |
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pubmed-article:20682682 | pubmed:year | 2010 | lld:pubmed |
pubmed-article:20682682 | pubmed:articleTitle | Central nervous system delivery of the antipsychotic olanzapine induces hepatic insulin resistance. | lld:pubmed |
pubmed-article:20682682 | pubmed:affiliation | Obesity Research Center, University of Cincinnati, Cincinnati, Ohio, USA. | lld:pubmed |
pubmed-article:20682682 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:20682682 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:20682682 | lld:pubmed |