| pubmed-article:20676103 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:20676103 | lifeskim:mentions | umls-concept:C0014544 | lld:lifeskim |
| pubmed-article:20676103 | lifeskim:mentions | umls-concept:C0019564 | lld:lifeskim |
| pubmed-article:20676103 | lifeskim:mentions | umls-concept:C0015127 | lld:lifeskim |
| pubmed-article:20676103 | lifeskim:mentions | umls-concept:C0032824 | lld:lifeskim |
| pubmed-article:20676103 | lifeskim:mentions | umls-concept:C0856984 | lld:lifeskim |
| pubmed-article:20676103 | lifeskim:mentions | umls-concept:C1314792 | lld:lifeskim |
| pubmed-article:20676103 | lifeskim:mentions | umls-concept:C0814034 | lld:lifeskim |
| pubmed-article:20676103 | lifeskim:mentions | umls-concept:C1442161 | lld:lifeskim |
| pubmed-article:20676103 | lifeskim:mentions | umls-concept:C1416573 | lld:lifeskim |
| pubmed-article:20676103 | pubmed:issue | 9 | lld:pubmed |
| pubmed-article:20676103 | pubmed:dateCreated | 2010-8-26 | lld:pubmed |
| pubmed-article:20676103 | pubmed:abstractText | We found the voltage-gated K+ channel Kv12.2 to be a potent regulator of excitability in hippocampal pyramidal neurons. Genetic deletion and pharmacologic block of Kv12.2 substantially reduced the firing threshold of these neurons. Kv12.2-/- (also known as Kcnh3-/-) mice showed signs of persistent neuronal hyperexcitability including frequent interictal spiking, spontaneous seizures and increased sensitivity to the chemoconvulsant pentylenetetrazol. | lld:pubmed |
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| pubmed-article:20676103 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20676103 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:20676103 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20676103 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20676103 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20676103 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20676103 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20676103 | pubmed:language | eng | lld:pubmed |
| pubmed-article:20676103 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20676103 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:20676103 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20676103 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20676103 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20676103 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20676103 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:20676103 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:20676103 | pubmed:issn | 1546-1726 | lld:pubmed |
| pubmed-article:20676103 | pubmed:author | pubmed-author:TanL BLB | lld:pubmed |
| pubmed-article:20676103 | pubmed:author | pubmed-author:BertasoFederi... | lld:pubmed |
| pubmed-article:20676103 | pubmed:author | pubmed-author:ZhangXiaofeiX | lld:pubmed |
| pubmed-article:20676103 | pubmed:author | pubmed-author:SchmedtChrist... | lld:pubmed |
| pubmed-article:20676103 | pubmed:author | pubmed-author:NoebelsJeffre... | lld:pubmed |
| pubmed-article:20676103 | pubmed:author | pubmed-author:AvisJacquelin... | lld:pubmed |
| pubmed-article:20676103 | pubmed:author | pubmed-author:BaumgärtelKar... | lld:pubmed |
| pubmed-article:20676103 | pubmed:author | pubmed-author:ClancySinead... | lld:pubmed |
| pubmed-article:20676103 | pubmed:author | pubmed-author:YooJong WJW | lld:pubmed |
| pubmed-article:20676103 | pubmed:author | pubmed-author:JeglaTimothyT | lld:pubmed |
| pubmed-article:20676103 | pubmed:author | pubmed-author:DaguiaCatheri... | lld:pubmed |
| pubmed-article:20676103 | pubmed:author | pubmed-author:CienfuegosCyn... | lld:pubmed |
| pubmed-article:20676103 | pubmed:author | pubmed-author:WilmotCarlyC | lld:pubmed |
| pubmed-article:20676103 | pubmed:author | pubmed-author:HunyhTrucT | lld:pubmed |
| pubmed-article:20676103 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:20676103 | pubmed:volume | 13 | lld:pubmed |
| pubmed-article:20676103 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:20676103 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:20676103 | pubmed:pagination | 1056-8 | lld:pubmed |
| pubmed-article:20676103 | pubmed:dateRevised | 2011-7-25 | lld:pubmed |
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| pubmed-article:20676103 | pubmed:meshHeading | pubmed-meshheading:20676103... | lld:pubmed |
| pubmed-article:20676103 | pubmed:year | 2010 | lld:pubmed |
| pubmed-article:20676103 | pubmed:articleTitle | Deletion of the potassium channel Kv12.2 causes hippocampal hyperexcitability and epilepsy. | lld:pubmed |
| pubmed-article:20676103 | pubmed:affiliation | Department of Cell Biology, The Scripps Research Institute, La Jolla, California, USA. | lld:pubmed |
| pubmed-article:20676103 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:20676103 | pubmed:publicationType | In Vitro | lld:pubmed |
| pubmed-article:20676103 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| pubmed-article:20676103 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
| entrez-gene:16512 | entrezgene:pubmed | pubmed-article:20676103 | lld:entrezgene |
| http://linkedlifedata.com/r... | entrezgene:pubmed | pubmed-article:20676103 | lld:entrezgene |
