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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
11
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pubmed:dateCreated |
2010-11-2
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pubmed:abstractText |
Mast cells are tissue-resident immune sentinels that are implicated in the pathogenesis of inflammatory joint disease. The aim of this study was to test our hypothesis that complement fragments could be key activators of synovial mast cells in autoimmune arthritis.
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pubmed:grant |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Nov
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pubmed:issn |
1529-0131
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pubmed:author |
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pubmed:copyrightInfo |
Copyright © 2010 by the American College of Rheumatology.
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pubmed:issnType |
Electronic
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pubmed:volume |
62
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
3322-33
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pubmed:dateRevised |
2011-11-1
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pubmed:meshHeading |
pubmed-meshheading:20662064-Animals,
pubmed-meshheading:20662064-Antigen-Antibody Complex,
pubmed-meshheading:20662064-Arthritis, Experimental,
pubmed-meshheading:20662064-Cells, Cultured,
pubmed-meshheading:20662064-Mast Cells,
pubmed-meshheading:20662064-Mice,
pubmed-meshheading:20662064-Neutrophil Infiltration,
pubmed-meshheading:20662064-Receptor, Anaphylatoxin C5a,
pubmed-meshheading:20662064-Receptors, IgG,
pubmed-meshheading:20662064-Reverse Transcriptase Polymerase Chain Reaction
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pubmed:year |
2010
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pubmed:articleTitle |
C5a receptor enables participation of mast cells in immune complex arthritis independently of Fc? receptor modulation.
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pubmed:affiliation |
Brigham and Women's Hospital and Children's Hospital Boston, Boston, Massachusetts, USA.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't,
Research Support, N.I.H., Extramural
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