Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
10
pubmed:dateCreated
2010-9-16
pubmed:abstractText
Vascular calcification is an important risk factor for cardiovascular diseases. Here, we investigated a role of dedifferentiated vascular smooth muscle cells (VSMCs) in the atherosclerotic intimal calcification.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Oct
pubmed:issn
1524-4636
pubmed:author
pubmed:issnType
Electronic
pubmed:volume
30
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
1908-15
pubmed:meshHeading
pubmed-meshheading:20651281-Animals, pubmed-meshheading:20651281-Apolipoproteins E, pubmed-meshheading:20651281-Atherosclerosis, pubmed-meshheading:20651281-Bone Morphogenetic Protein 2, pubmed-meshheading:20651281-Calcinosis, pubmed-meshheading:20651281-Cell Dedifferentiation, pubmed-meshheading:20651281-Cell Differentiation, pubmed-meshheading:20651281-Cells, Cultured, pubmed-meshheading:20651281-Culture Media, Conditioned, pubmed-meshheading:20651281-Female, pubmed-meshheading:20651281-Gene Expression, pubmed-meshheading:20651281-Humans, pubmed-meshheading:20651281-Male, pubmed-meshheading:20651281-Mice, pubmed-meshheading:20651281-Mice, Inbred C57BL, pubmed-meshheading:20651281-Mice, Knockout, pubmed-meshheading:20651281-Mice, Transgenic, pubmed-meshheading:20651281-Models, Biological, pubmed-meshheading:20651281-Myocytes, Smooth Muscle, pubmed-meshheading:20651281-Osteoblasts, pubmed-meshheading:20651281-Osteogenesis, pubmed-meshheading:20651281-Paracrine Communication, pubmed-meshheading:20651281-Tunica Intima
pubmed:year
2010
pubmed:articleTitle
Paracrine osteogenic signals via bone morphogenetic protein-2 accelerate the atherosclerotic intimal calcification in vivo.
pubmed:affiliation
Department of Cardiovascular Medicine, Kyoto Prefectural University School of Medicine, Kamigyo, Kyoto, Japan.
pubmed:publicationType
Journal Article, Research Support, Non-U.S. Gov't