|
rdf:type |
|
|
lifeskim:mentions |
umls-concept:C0006560,
umls-concept:C0016884,
umls-concept:C0018787,
umls-concept:C0026473,
umls-concept:C0030705,
umls-concept:C0041904,
umls-concept:C0264716,
umls-concept:C0439849,
umls-concept:C0441471,
umls-concept:C0445223,
umls-concept:C1327413,
umls-concept:C1522564,
umls-concept:C1552599,
umls-concept:C1704787,
umls-concept:C1883709
|
|
pubmed:issue |
7
|
|
pubmed:dateCreated |
2010-7-8
|
|
pubmed:abstractText |
An elevation of the cardiac troponin T (TnT) level identifies patients with ongoing myocardial damage (OMD) and at increased risk for future cardiac events in chronic heart failure (CHF). C-reactive protein (CRP) upregulates monocyte proinflammatory cytokine production and this upregulation appears to play an important role on OMD.
|
|
pubmed:language |
eng
|
|
pubmed:journal |
|
|
pubmed:citationSubset |
IM
|
|
pubmed:chemical |
|
|
pubmed:status |
MEDLINE
|
|
pubmed:month |
Jul
|
|
pubmed:issn |
1532-8414
|
|
pubmed:author |
|
|
pubmed:copyrightInfo |
Copyright 2010 Elsevier Inc. All rights reserved.
|
|
pubmed:issnType |
Electronic
|
|
pubmed:volume |
16
|
|
pubmed:owner |
NLM
|
|
pubmed:authorsComplete |
Y
|
|
pubmed:pagination |
562-71
|
|
pubmed:meshHeading |
pubmed-meshheading:20610232-Aged,
pubmed-meshheading:20610232-C-Reactive Protein,
pubmed-meshheading:20610232-Cytokines,
pubmed-meshheading:20610232-Female,
pubmed-meshheading:20610232-Follow-Up Studies,
pubmed-meshheading:20610232-Heart Failure,
pubmed-meshheading:20610232-Humans,
pubmed-meshheading:20610232-Leukocytes, Mononuclear,
pubmed-meshheading:20610232-Male,
pubmed-meshheading:20610232-Middle Aged,
pubmed-meshheading:20610232-Monocytes,
pubmed-meshheading:20610232-Myocardium,
pubmed-meshheading:20610232-Up-Regulation
|
|
pubmed:year |
2010
|
|
pubmed:articleTitle |
Upregulation of monocyte proinflammatory cytokine production by C-reactive protein is significantly related to ongoing myocardial damage and future cardiac events in patients with chronic heart failure.
|
|
pubmed:affiliation |
Department of Cardiology, Tama-Nagayama Hospital, Nippon Medical School, Tokyo, Japan. Nakagomi@nms.ac.jp
|
|
pubmed:publicationType |
Journal Article,
Comparative Study
|
