20541697

Source:http://linkedlifedata.com/resource/pubmed/id/20541697

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Predicate	Object
rdf:type	pubmed:Citation
lifeskim:mentions	umls-concept:C0023418, umls-concept:C0032172, umls-concept:C0033414, umls-concept:C0033684, umls-concept:C0235480, umls-concept:C0279389
pubmed:issue	6
pubmed:dateCreated	2010-6-14
pubmed:abstractText	MLL1 fusions are among the most potent oncogenic drivers of leukemia development. In recent articles published in Molecular Cell and in Cancer Cell, researchers find that MLL1 fusions are reliant on a physical interaction with the PAF transcription elongation complex for their recruitment to chromatin and, consequently, leukemic transformation.
pubmed:language	eng
pubmed:journal	http://linkedlifedata.com/resource/pubmed/journal/101130617
pubmed:status	PubMed-not-MEDLINE
pubmed:month	Jun
pubmed:issn	1878-3686
pubmed:author	pubmed-author:SpeckNancy ANA, pubmed-author:VakocChristopher RCR
pubmed:copyrightInfo	Copyright 2010 Elsevier Inc. All rights reserved.
pubmed:issnType	Electronic
pubmed:day	15
pubmed:volume	17
pubmed:owner	NLM
pubmed:authorsComplete	Y
pubmed:pagination	531-2
pubmed:year	2010
pubmed:articleTitle	PAF is in the cabal of MLL1-interacting proteins that promote leukemia.
pubmed:affiliation	Abramson Family Cancer Research Institute and Department of Cell and Developmental Biology, University of Pennsylvania, Philadelphia, PA 19104, USA.
pubmed:publicationType	Journal Article