20541486

Source:http://linkedlifedata.com/resource/pubmed/id/20541486

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Predicate	Object
rdf:type	pubmed:Citation
lifeskim:mentions	umls-concept:C0026882, umls-concept:C0079419, umls-concept:C0079488, umls-concept:C0278701, umls-concept:C0598086, umls-concept:C0598388, umls-concept:C1415887, umls-concept:C1419040, umls-concept:C1420433, umls-concept:C1424666, umls-concept:C1707520
pubmed:issue	5
pubmed:dateCreated	2010-10-4
pubmed:abstractText	Helicobacter pylori infection is an established risk factor for gastric cancer development, but the exact underlying mechanism still remains obscure. The inactivation of tumor suppressor genes such as p53 and p27(KIP1) is a hypothesized mechanism, although there is no consensus regarding the influence of H. pylori cagA(+) in the development of these genetic alterations.
pubmed:language	eng
pubmed:journal	http://linkedlifedata.com/resource/pubmed/journal/101508793
pubmed:citationSubset	IM
pubmed:chemical	http://linkedlifedata.com/resource/pubmed/chemical/Antigens, Bacterial, http://linkedlifedata.com/resource/pubmed/chemical/Bacterial Proteins, http://linkedlifedata.com/resource/pubmed/chemical/CDKN1B protein, human, http://linkedlifedata.com/resource/pubmed/chemical/Cyclin-Dependent Kinase Inhibitor..., http://linkedlifedata.com/resource/pubmed/chemical/Intracellular Signaling Peptides..., http://linkedlifedata.com/resource/pubmed/chemical/cagA protein, Helicobacter pylori
pubmed:status	MEDLINE
pubmed:month	Oct
pubmed:issn	1877-783X
pubmed:author	pubmed-author:AndréAngela RosaAR, pubmed-author:FerrasiAdriana CamargoAC, pubmed-author:FerreiraMárcia Valéria PitombeiraMV, pubmed-author:MotaRosa Maria SalaniRM, pubmed-author:PardiniMaria Inês de Moura CamposMI, pubmed-author:RabenhorstSílvia Helena BaremSH
pubmed:copyrightInfo	Copyright © 2010 Elsevier Ltd. All rights reserved.
pubmed:issnType	Electronic
pubmed:volume	34
pubmed:owner	NLM
pubmed:authorsComplete	Y
pubmed:pagination	618-25
pubmed:dateRevised	2011-11-17
pubmed:meshHeading	pubmed-meshheading:20541486-Adenocarcinoma, pubmed-meshheading:20541486-Adult, pubmed-meshheading:20541486-Aged, pubmed-meshheading:20541486-Aged, 80 and over, pubmed-meshheading:20541486-Antigens, Bacterial, pubmed-meshheading:20541486-Bacterial Proteins, pubmed-meshheading:20541486-Cyclin-Dependent Kinase Inhibitor p27, pubmed-meshheading:20541486-Female, pubmed-meshheading:20541486-Gene Silencing, pubmed-meshheading:20541486-Genes, p53, pubmed-meshheading:20541486-Helicobacter Infections, pubmed-meshheading:20541486-Helicobacter pylori, pubmed-meshheading:20541486-Humans, pubmed-meshheading:20541486-Immunohistochemistry, pubmed-meshheading:20541486-Intracellular Signaling Peptides and Proteins, pubmed-meshheading:20541486-Male, pubmed-meshheading:20541486-Middle Aged, pubmed-meshheading:20541486-Mutation, pubmed-meshheading:20541486-Polymerase Chain Reaction, pubmed-meshheading:20541486-Polymorphism, Single-Stranded Conformational, pubmed-meshheading:20541486-Stomach Neoplasms, pubmed-meshheading:20541486-Young Adult
pubmed:year	2010
pubmed:articleTitle	Gastric adenocarcinoma and Helicobacter pylori: correlation with p53 mutation and p27 immunoexpression.
pubmed:affiliation	Molecular Genetics Laboratory, Department of Pathology and Forensic Medicine, Federal University of Ceará, Rua Alexandre Baraúna, 949, Rodolfo Téofilo, CEP 60430-160, Fortaleza-CE, Brazil. angelarandre@yahoo.com.br
pubmed:publicationType	Journal Article