20522804

Source:http://linkedlifedata.com/resource/pubmed/id/20522804

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Predicate	Object
rdf:type	pubmed:Citation
lifeskim:mentions	umls-concept:C0016030, umls-concept:C0027051, umls-concept:C0031437, umls-concept:C0037083, umls-concept:C0043240, umls-concept:C0542341, umls-concept:C0851285, umls-concept:C0919432, umls-concept:C1710082
pubmed:issue	3
pubmed:dateCreated	2010-8-6
pubmed:abstractText	Cardiac fibroblasts are key effector cells in the pathogenesis of cardiac fibrosis. Transforming growth factor (TGF)-beta/Smad3 signaling is activated in the border zone of healing infarcts and induces fibrotic remodeling of the infarcted ventricle contributing to the development of diastolic dysfunction.
pubmed:grant	http://linkedlifedata.com/resource/pubmed/grant/R01 HL-76246, http://linkedlifedata.com/resource/pubmed/grant/R01 HL-85440, http://linkedlifedata.com/resource/pubmed/grant/R01 HL076246-05, http://linkedlifedata.com/resource/pubmed/grant/R01 HL076246-06, http://linkedlifedata.com/resource/pubmed/grant/R01 HL085440-03
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pubmed:language	eng
pubmed:journal	http://linkedlifedata.com/resource/pubmed/journal/0047103
pubmed:citationSubset	IM
pubmed:chemical	http://linkedlifedata.com/resource/pubmed/chemical/Extracellular Matrix Proteins, http://linkedlifedata.com/resource/pubmed/chemical/Smad3 Protein, http://linkedlifedata.com/resource/pubmed/chemical/Smad3 protein, mouse, http://linkedlifedata.com/resource/pubmed/chemical/Trans-Activators, http://linkedlifedata.com/resource/pubmed/chemical/Transforming Growth Factor beta1
pubmed:status	MEDLINE
pubmed:month	Aug
pubmed:issn	1524-4571
pubmed:author	pubmed-author:BujakMarcinM, pubmed-author:DobaczewskiMarcinM, pubmed-author:FrangogiannisNikolaos GNG, pubmed-author:Gonzalez-QuesadaCarlosC, pubmed-author:MendozaLeonardo HLH, pubmed-author:N, pubmed-author:WangXiao-FanXF
pubmed:issnType	Electronic
pubmed:day	6
pubmed:volume	107
pubmed:owner	NLM
pubmed:authorsComplete	Y
pubmed:pagination	418-28
pubmed:dateRevised	2011-9-26
pubmed:meshHeading	pubmed-meshheading:20522804-Animals, pubmed-meshheading:20522804-Cell Differentiation, pubmed-meshheading:20522804-Cell Division, pubmed-meshheading:20522804-Cell Movement, pubmed-meshheading:20522804-Extracellular Matrix Proteins, pubmed-meshheading:20522804-Fibroblasts, pubmed-meshheading:20522804-Mice, pubmed-meshheading:20522804-Mice, Knockout, pubmed-meshheading:20522804-Myocardial Contraction, pubmed-meshheading:20522804-Myocardial Infarction, pubmed-meshheading:20522804-Phenotype, pubmed-meshheading:20522804-Smad3 Protein, pubmed-meshheading:20522804-Trans-Activators, pubmed-meshheading:20522804-Transforming Growth Factor beta1, pubmed-meshheading:20522804-Up-Regulation, pubmed-meshheading:20522804-Wound Healing
pubmed:year	2010
pubmed:articleTitle	Smad3 signaling critically regulates fibroblast phenotype and function in healing myocardial infarction.

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