| pubmed-article:20497634 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:20497634 | lifeskim:mentions | umls-concept:C0241888 | lld:lifeskim |
| pubmed-article:20497634 | lifeskim:mentions | umls-concept:C0031046 | lld:lifeskim |
| pubmed-article:20497634 | lifeskim:mentions | umls-concept:C1456820 | lld:lifeskim |
| pubmed-article:20497634 | lifeskim:mentions | umls-concept:C0039082 | lld:lifeskim |
| pubmed-article:20497634 | lifeskim:mentions | umls-concept:C0034897 | lld:lifeskim |
| pubmed-article:20497634 | lifeskim:mentions | umls-concept:C0009085 | lld:lifeskim |
| pubmed-article:20497634 | lifeskim:mentions | umls-concept:C0332182 | lld:lifeskim |
| pubmed-article:20497634 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:20497634 | pubmed:dateCreated | 2010-6-25 | lld:pubmed |
| pubmed-article:20497634 | pubmed:abstractText | Although several causes of recurrent pericarditis have been identified, the etiology remains obscure in most cases. The tumour necrosis factor receptor-1 associated periodic syndrome (TRAPS) is the most common autosomal dominant autoinflammatory disorder and is caused by mutations in the TNFRSF1A gene encoding the 55-kD receptor for tumour necrosis factor-(TNF)-alpha. Serosal membrane inflammation is a common feature of TRAPS, usually in the form of polyserositis. In addition, patients affected with recurrent pericarditis as the only clinical manifestation of TRAPS have been recently described. Our aim was to investigate the possible involvement of mutations in the TNFRSF1A gene in a cohort of patients affected with idiopathic recurrent pericarditis. | lld:pubmed |
| pubmed-article:20497634 | pubmed:language | eng | lld:pubmed |
| pubmed-article:20497634 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20497634 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:20497634 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20497634 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20497634 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:20497634 | pubmed:issn | 0392-856X | lld:pubmed |
| pubmed-article:20497634 | pubmed:author | pubmed-author:GaleazziMauro... | lld:pubmed |
| pubmed-article:20497634 | pubmed:author | pubmed-author:Laghi... | lld:pubmed |
| pubmed-article:20497634 | pubmed:author | pubmed-author:CantariniLuca... | lld:pubmed |
| pubmed-article:20497634 | pubmed:author | pubmed-author:LucheriniOrso... | lld:pubmed |
| pubmed-article:20497634 | pubmed:author | pubmed-author:BaldariCosima... | lld:pubmed |
| pubmed-article:20497634 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:20497634 | pubmed:volume | 28 | lld:pubmed |
| pubmed-article:20497634 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:20497634 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:20497634 | pubmed:pagination | 405-7 | lld:pubmed |
| pubmed-article:20497634 | pubmed:meshHeading | pubmed-meshheading:20497634... | lld:pubmed |
| pubmed-article:20497634 | pubmed:meshHeading | pubmed-meshheading:20497634... | lld:pubmed |
| pubmed-article:20497634 | pubmed:meshHeading | pubmed-meshheading:20497634... | lld:pubmed |
| pubmed-article:20497634 | pubmed:meshHeading | pubmed-meshheading:20497634... | lld:pubmed |
| pubmed-article:20497634 | pubmed:meshHeading | pubmed-meshheading:20497634... | lld:pubmed |
| pubmed-article:20497634 | pubmed:meshHeading | pubmed-meshheading:20497634... | lld:pubmed |
| pubmed-article:20497634 | pubmed:meshHeading | pubmed-meshheading:20497634... | lld:pubmed |
| pubmed-article:20497634 | pubmed:meshHeading | pubmed-meshheading:20497634... | lld:pubmed |
| pubmed-article:20497634 | pubmed:meshHeading | pubmed-meshheading:20497634... | lld:pubmed |
| pubmed-article:20497634 | pubmed:meshHeading | pubmed-meshheading:20497634... | lld:pubmed |
| pubmed-article:20497634 | pubmed:meshHeading | pubmed-meshheading:20497634... | lld:pubmed |
| pubmed-article:20497634 | pubmed:meshHeading | pubmed-meshheading:20497634... | lld:pubmed |
| pubmed-article:20497634 | pubmed:meshHeading | pubmed-meshheading:20497634... | lld:pubmed |
| pubmed-article:20497634 | pubmed:meshHeading | pubmed-meshheading:20497634... | lld:pubmed |
| pubmed-article:20497634 | pubmed:meshHeading | pubmed-meshheading:20497634... | lld:pubmed |
| pubmed-article:20497634 | pubmed:articleTitle | Familial clustering of recurrent pericarditis may disclose tumour necrosis factor receptor-associated periodic syndrome. | lld:pubmed |
| pubmed-article:20497634 | pubmed:affiliation | Interdepartmental Research Center of Systemic Autoimmune and Autoinflammatory Diseases, Unit of Rheumatology, Policlinico Le Scotte, University of Siena, Siena, Italy. cantariniluca@hotmail.com | lld:pubmed |
| pubmed-article:20497634 | pubmed:publicationType | Journal Article | lld:pubmed |
| entrez-gene:7132 | entrezgene:pubmed | pubmed-article:20497634 | lld:entrezgene |
| http://linkedlifedata.com/r... | entrezgene:pubmed | pubmed-article:20497634 | lld:entrezgene |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:20497634 | lld:pubmed |
