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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
1
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pubmed:dateCreated |
2010-7-9
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pubmed:abstractText |
Unrepaired cardiomyocyte membrane injury causes irreplaceable cell loss, leading to myocardial fibrosis and eventually heart failure. However, the cellular and molecular mechanisms of cardiac membrane repair are largely unknown. MG53, a newly identified striated muscle-specific protein, is involved in skeletal muscle membrane repair. But the role of MG53 in the heart has not been determined.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Jul
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pubmed:issn |
1524-4571
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pubmed:author |
pubmed-author:ChengHepingH,
pubmed-author:HanPeidongP,
pubmed-author:JiGuangjuG,
pubmed-author:LiangHanH,
pubmed-author:LinPeihuiP,
pubmed-author:MaJianjieJ,
pubmed-author:TakeshimaHiroshiH,
pubmed-author:WangXianhuaX,
pubmed-author:WangYanruY,
pubmed-author:WeislederNoahN,
pubmed-author:XiaoRui-PingRP,
pubmed-author:XieWenjunW,
pubmed-author:ZhangYiY,
pubmed-author:ZhengChenC,
pubmed-author:ZhengMingM
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pubmed:issnType |
Electronic
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pubmed:day |
9
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pubmed:volume |
107
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
76-83
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pubmed:meshHeading |
|
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pubmed:year |
2010
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pubmed:articleTitle |
Cardioprotection of ischemia/reperfusion injury by cholesterol-dependent MG53-mediated membrane repair.
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pubmed:affiliation |
Institute of Molecular Medicine, State Key Laboratory of Biomembrane and Membrane Biotechnology, Peking University, Beijing, China. xianhua@pku.edu.cn
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pubmed:publicationType |
Journal Article,
Comparative Study,
Research Support, Non-U.S. Gov't
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