Linked Life Data

20456770

Source:http://linkedlifedata.com/resource/pubmed/id/20456770

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:dateCreated
2010-5-11
pubmed:abstractText
Chronic lung damage (bronchopulmonary dysplasia (BPD)) is one of the most serious complications affecting preterm neonates. During the last decade the aetiology of BPD has changed. Whereas 'classic BPD' was characterised mainly by lung damage and fibrosis caused by oxygen toxicity and mechanical ventilation, 'new BPD' is characterised by a disorder in lung development. This aetiological shift has been brought about by improved survival in extremely premature infants as a result of, for instance, antenatal corticosteroid administration and postnatal surfactant therapy. New BPD requires a new therapeutic approach. Therapeutic options for developing BPD include caffeine, vitamin A and postnatal corticosteroids. Once BPD has occurred, diuretics and inhaled bronchodilators and corticosteroids may be useful. However, the available therapies decrease the risk of developing BPD by just a small percent. In the future, artificial surfactants and non-invasive ventilation may prove to be useful in the prevention of BPD.
pubmed:language
dut
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:issn
1876-8784
pubmed:author
pubmed:issnType
Electronic
pubmed:volume
154
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
A1024
pubmed:dateRevised
2010-11-4
pubmed:meshHeading
pubmed:year
2010
pubmed:articleTitle
[From classic to new bronchopulmonary dysplasia].
pubmed:affiliation
Universitair Medisch Centrum, Maastricht, afd. Kindergeneeskunde, School for Oncology and Developmental Biology (GROW), Maastricht, The Netherlands.
pubmed:publicationType
Journal Article, English Abstract, Review