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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
2
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pubmed:dateCreated |
2010-5-10
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pubmed:abstractText |
The regulation of Ca(2+) entry and removal is a fine-tuned process which remains not well understood in mouse retinal ganglion cells (RGCs). The latter are known to be sensitive to dysfunctions of mitochondria, organelles playing a pivotal role in Ca(2+) reuptake.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
http://linkedlifedata.com/resource/pubmed/chemical/Calcium,
http://linkedlifedata.com/resource/pubmed/chemical/Calcium Channel Blockers,
http://linkedlifedata.com/resource/pubmed/chemical/Calcium-Transporting ATPases,
http://linkedlifedata.com/resource/pubmed/chemical/Fura-2,
http://linkedlifedata.com/resource/pubmed/chemical/GABA-A Receptor Antagonists,
http://linkedlifedata.com/resource/pubmed/chemical/GTP Phosphohydrolases,
http://linkedlifedata.com/resource/pubmed/chemical/Indoles,
http://linkedlifedata.com/resource/pubmed/chemical/Opa1 protein (GTPase), mouse,
http://linkedlifedata.com/resource/pubmed/chemical/Potassium,
http://linkedlifedata.com/resource/pubmed/chemical/RNA, Small Interfering,
http://linkedlifedata.com/resource/pubmed/chemical/Receptors, AMPA,
http://linkedlifedata.com/resource/pubmed/chemical/Receptors, GABA-A,
http://linkedlifedata.com/resource/pubmed/chemical/Receptors, N-Methyl-D-Aspartate,
http://linkedlifedata.com/resource/pubmed/chemical/cyclopiazonic acid
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pubmed:status |
MEDLINE
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pubmed:month |
Jun
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pubmed:issn |
1744-5094
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pubmed:author |
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pubmed:issnType |
Electronic
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pubmed:volume |
31
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
53-65
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pubmed:dateRevised |
2010-11-18
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pubmed:meshHeading |
pubmed-meshheading:20450306-Animals,
pubmed-meshheading:20450306-Animals, Newborn,
pubmed-meshheading:20450306-Blotting, Western,
pubmed-meshheading:20450306-Calcium,
pubmed-meshheading:20450306-Calcium Channel Blockers,
pubmed-meshheading:20450306-Calcium Signaling,
pubmed-meshheading:20450306-Calcium-Transporting ATPases,
pubmed-meshheading:20450306-Cell Survival,
pubmed-meshheading:20450306-Cells, Cultured,
pubmed-meshheading:20450306-Cytosol,
pubmed-meshheading:20450306-Fura-2,
pubmed-meshheading:20450306-GABA-A Receptor Antagonists,
pubmed-meshheading:20450306-GTP Phosphohydrolases,
pubmed-meshheading:20450306-Homeostasis,
pubmed-meshheading:20450306-Indoles,
pubmed-meshheading:20450306-Mice,
pubmed-meshheading:20450306-Mice, Inbred C57BL,
pubmed-meshheading:20450306-Microscopy, Confocal,
pubmed-meshheading:20450306-Mitochondria,
pubmed-meshheading:20450306-Potassium,
pubmed-meshheading:20450306-RNA, Small Interfering,
pubmed-meshheading:20450306-Receptors, AMPA,
pubmed-meshheading:20450306-Receptors, GABA-A,
pubmed-meshheading:20450306-Receptors, N-Methyl-D-Aspartate,
pubmed-meshheading:20450306-Retinal Ganglion Cells,
pubmed-meshheading:20450306-Transfection
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pubmed:year |
2010
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pubmed:articleTitle |
Characterization of Ca2+ signalling in postnatal mouse retinal ganglion cells: involvement of OPA1 in Ca2+ clearance.
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pubmed:affiliation |
Institut des Neurosciences de Montpellier, INSERM U583, Montpellier, France.
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pubmed:publicationType |
Journal Article
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