pubmed-article:20398276 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:20398276 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
pubmed-article:20398276 | lifeskim:mentions | umls-concept:C1513095 | lld:lifeskim |
pubmed-article:20398276 | lifeskim:mentions | umls-concept:C1171362 | lld:lifeskim |
pubmed-article:20398276 | lifeskim:mentions | umls-concept:C0021747 | lld:lifeskim |
pubmed-article:20398276 | lifeskim:mentions | umls-concept:C1564682 | lld:lifeskim |
pubmed-article:20398276 | lifeskim:mentions | umls-concept:C1709059 | lld:lifeskim |
pubmed-article:20398276 | pubmed:dateCreated | 2010-4-23 | lld:pubmed |
pubmed-article:20398276 | pubmed:abstractText | Endogenously produced interferons can regulate the growth of melanoma cells and are administered exogenously as therapeutic agents to patients with advanced cancer. We investigated the role of negative regulators of interferon signaling known as suppressors of cytokine signaling (SOCS) in mediating interferon-resistance in human melanoma cells. | lld:pubmed |
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pubmed-article:20398276 | pubmed:language | eng | lld:pubmed |
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pubmed-article:20398276 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:20398276 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:20398276 | pubmed:issn | 1471-2407 | lld:pubmed |
pubmed-article:20398276 | pubmed:author | pubmed-author:BecknellBrian... | lld:pubmed |
pubmed-article:20398276 | pubmed:author | pubmed-author:CarsonWilliam... | lld:pubmed |
pubmed-article:20398276 | pubmed:author | pubmed-author:YoungGregory... | lld:pubmed |
pubmed-article:20398276 | pubmed:author | pubmed-author:LesinskiGrego... | lld:pubmed |
pubmed-article:20398276 | pubmed:author | pubmed-author:BillMatthew... | lld:pubmed |
pubmed-article:20398276 | pubmed:author | pubmed-author:ZimmererJason... | lld:pubmed |
pubmed-article:20398276 | pubmed:author | pubmed-author:TrefryJohnJ | lld:pubmed |
pubmed-article:20398276 | pubmed:author | pubmed-author:KreinerMelani... | lld:pubmed |
pubmed-article:20398276 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:20398276 | pubmed:volume | 10 | lld:pubmed |
pubmed-article:20398276 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:20398276 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:20398276 | pubmed:pagination | 142 | lld:pubmed |
pubmed-article:20398276 | pubmed:dateRevised | 2011-11-17 | lld:pubmed |
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pubmed-article:20398276 | pubmed:meshHeading | pubmed-meshheading:20398276... | lld:pubmed |
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pubmed-article:20398276 | pubmed:meshHeading | pubmed-meshheading:20398276... | lld:pubmed |
pubmed-article:20398276 | pubmed:year | 2010 | lld:pubmed |
pubmed-article:20398276 | pubmed:articleTitle | Modulation of SOCS protein expression influences the interferon responsiveness of human melanoma cells. | lld:pubmed |
pubmed-article:20398276 | pubmed:affiliation | Department of Surgery Arthur G, James Cancer Hospital and Richard J, Solove Research Institute, The Ohio State University, Columbus, OH 43210, USA. | lld:pubmed |