rdf:type |
|
lifeskim:mentions |
|
pubmed:issue |
5
|
pubmed:dateCreated |
2010-5-7
|
pubmed:abstractText |
Because bepridil blocks multiple myocardial ionic channels, including the muscarinic acetylcholine receptor-operated potassium current (I(KAch)), bepridil is expected to suppress atrial fibrillation (AF) mediated by vagal nerve stimulation (VNS).
|
pubmed:language |
eng
|
pubmed:journal |
|
pubmed:citationSubset |
IM
|
pubmed:chemical |
|
pubmed:status |
MEDLINE
|
pubmed:month |
May
|
pubmed:issn |
1347-4820
|
pubmed:author |
|
pubmed:issnType |
Electronic
|
pubmed:volume |
74
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
895-902
|
pubmed:dateRevised |
2011-2-18
|
pubmed:meshHeading |
|
pubmed:year |
2010
|
pubmed:articleTitle |
Effect of bepridil in atrial fibrillation inducibility facilitated by vagal nerve stimulation. - Prevention of vagal nerve activation-induced shortening of the atrial action potential duration -.
|
pubmed:affiliation |
The First Department of Internal Medicine, Niigata University School of Medicine, Niigata, Japan.
|
pubmed:publicationType |
Journal Article
|