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20368219
Source:
http://linkedlifedata.com/resource/pubmed/id/20368219
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Statements in which the resource exists as a subject.
Predicate
Object
rdf:type
pubmed:Citation
lifeskim:mentions
umls-concept:C0003483
,
umls-concept:C0026809
,
umls-concept:C0038435
,
umls-concept:C0205225
,
umls-concept:C0225336
,
umls-concept:C0332307
,
umls-concept:C0333348
,
umls-concept:C0348013
pubmed:issue
4
pubmed:dateCreated
2010-4-6
pubmed:abstractText
The progression of diabetes is associated with profound endothelial dysfunction. We tested the hypothesis that cellular stress would be detectable in ECs retrieved from arterial and venous vessels of diabetic mice.
pubmed:language
eng
pubmed:journal
http://linkedlifedata.com/resource/pubmed/journal/101234011
pubmed:citationSubset
IM
pubmed:chemical
http://linkedlifedata.com/resource/pubmed/chemical/Caspase 1
,
http://linkedlifedata.com/resource/pubmed/chemical/Inflammation Mediators
,
http://linkedlifedata.com/resource/pubmed/chemical/Interleukin-1beta
,
http://linkedlifedata.com/resource/pubmed/chemical/RNA, Messenger
pubmed:status
MEDLINE
pubmed:month
Oct
pubmed:issn
1752-8984
pubmed:author
pubmed-author:BucciarelliLoredana GLG
,
pubmed-author:ColomboPaolo CPC
,
pubmed-author:GandaAnjaliA
,
pubmed-author:HudsonBarry IBI
,
pubmed-author:KaleaAnastasia ZAZ
,
pubmed-author:KebschullMoritzM
,
pubmed-author:LallaEvanthiaE
,
pubmed-author:PollreiszAndreasA
,
pubmed-author:RamasamyRavichandranR
,
pubmed-author:SchmidtAnn MarieAM
,
pubmed-author:YanShi FangSF
,
pubmed-author:ZouYu ShanYS
pubmed:issnType
Electronic
pubmed:volume
6
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
249-61
pubmed:meshHeading
pubmed-meshheading:20368219-Animals
,
pubmed-meshheading:20368219-Aorta
,
pubmed-meshheading:20368219-Apoptosis
,
pubmed-meshheading:20368219-Caspase 1
,
pubmed-meshheading:20368219-Cell Separation
,
pubmed-meshheading:20368219-Cells, Cultured
,
pubmed-meshheading:20368219-Diabetes Mellitus, Experimental
,
pubmed-meshheading:20368219-Diabetes Mellitus, Type 1
,
pubmed-meshheading:20368219-Endothelial Cells
,
pubmed-meshheading:20368219-Gene Expression Profiling
,
pubmed-meshheading:20368219-Gene Expression Regulation
,
pubmed-meshheading:20368219-Inflammation
,
pubmed-meshheading:20368219-Inflammation Mediators
,
pubmed-meshheading:20368219-Interleukin-1beta
,
pubmed-meshheading:20368219-Male
,
pubmed-meshheading:20368219-Mice
,
pubmed-meshheading:20368219-Mice, Inbred C57BL
,
pubmed-meshheading:20368219-Phenotype
,
pubmed-meshheading:20368219-RNA, Messenger
,
pubmed-meshheading:20368219-Reverse Transcriptase Polymerase Chain Reaction
,
pubmed-meshheading:20368219-Stress, Physiological
,
pubmed-meshheading:20368219-Time Factors
,
pubmed-meshheading:20368219-Vena Cava, Inferior
pubmed:year
2009
pubmed:articleTitle
Inflammatory stress in primary venous and aortic endothelial cells of type 1 diabetic mice.
pubmed:affiliation
IRCCS Istituto Clinico Humanitas, Via Manzoni 56, Rozzano 20089, Milan, Italy.
pubmed:publicationType
Journal Article
,
Research Support, U.S. Gov't, P.H.S.
,
Research Support, Non-U.S. Gov't