Source:http://linkedlifedata.com/resource/pubmed/id/20224273
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
4
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pubmed:dateCreated |
2010-4-14
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pubmed:abstractText |
The role of nitric oxide (NO) is controversial in diabetes nephropathy progression and the mechanisms remain unknown, especially in non-obese type 2 diabetes. To examine mechanisms of nephropathy progression in non-obese type 2 diabetes, we used spontaneously diabetic Torii (SDT) rats, a newly established model of non-obese type 2 diabetes.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:issn |
1421-9670
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pubmed:author | |
pubmed:copyrightInfo |
2010 S. Karger AG, Basel.
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pubmed:issnType |
Electronic
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pubmed:volume |
31
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
342-52
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pubmed:meshHeading |
pubmed-meshheading:20224273-Animals,
pubmed-meshheading:20224273-Diabetes Mellitus, Type 2,
pubmed-meshheading:20224273-Diabetic Nephropathies,
pubmed-meshheading:20224273-Disease Progression,
pubmed-meshheading:20224273-Male,
pubmed-meshheading:20224273-Nitric Oxide,
pubmed-meshheading:20224273-Oxidative Stress,
pubmed-meshheading:20224273-Rats,
pubmed-meshheading:20224273-Rats, Sprague-Dawley
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pubmed:year |
2010
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pubmed:articleTitle |
Oxidative and nitrosative stress and progression of diabetic nephropathy in type 2 diabetes.
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pubmed:affiliation |
Division of Nephrology and Kidney Center, Kobe University School of Medicine, Japan.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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