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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
5
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pubmed:dateCreated |
2010-4-15
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pubmed:abstractText |
Enhanced endothelial permeability leading to intimal accumulation of low-density lipoproteins (LDL) stimulates the formation of atherosclerotic lesions. Histamine is known to increase vascular permeability. Whether this affects the formation of atherosclerotic lesions, however, remains elusive.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
http://linkedlifedata.com/resource/pubmed/chemical/Apolipoproteins E,
http://linkedlifedata.com/resource/pubmed/chemical/Histamine,
http://linkedlifedata.com/resource/pubmed/chemical/Histamine H1 Antagonists,
http://linkedlifedata.com/resource/pubmed/chemical/Histamine H2 Antagonists,
http://linkedlifedata.com/resource/pubmed/chemical/Inflammation Mediators,
http://linkedlifedata.com/resource/pubmed/chemical/Lipoproteins, LDL,
http://linkedlifedata.com/resource/pubmed/chemical/Receptors, Histamine H1,
http://linkedlifedata.com/resource/pubmed/chemical/Receptors, Histamine H2
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pubmed:status |
MEDLINE
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pubmed:month |
May
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pubmed:issn |
1524-4636
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pubmed:author |
pubmed-author:AkhmedovAlexanderA,
pubmed-author:BecherBurkhardB,
pubmed-author:BorénJanJ,
pubmed-author:HofmannJaninJ,
pubmed-author:JohansenPålP,
pubmed-author:LüscherThomas FTF,
pubmed-author:MocharlaPavaniP,
pubmed-author:RohrerLuciaL,
pubmed-author:RozenbergIzabelaI,
pubmed-author:SlukaSusanna H MSH,
pubmed-author:SolizJorgeJ,
pubmed-author:SteffelJanJ,
pubmed-author:TannerFelix CFC,
pubmed-author:WatanabeTakeshiT
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pubmed:issnType |
Electronic
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pubmed:volume |
30
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
923-30
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pubmed:meshHeading |
pubmed-meshheading:20203300-Animals,
pubmed-meshheading:20203300-Aortic Diseases,
pubmed-meshheading:20203300-Aortitis,
pubmed-meshheading:20203300-Apolipoproteins E,
pubmed-meshheading:20203300-Atherosclerosis,
pubmed-meshheading:20203300-Bone Marrow Transplantation,
pubmed-meshheading:20203300-Capillary Permeability,
pubmed-meshheading:20203300-Cell Proliferation,
pubmed-meshheading:20203300-Disease Models, Animal,
pubmed-meshheading:20203300-Histamine,
pubmed-meshheading:20203300-Histamine H1 Antagonists,
pubmed-meshheading:20203300-Histamine H2 Antagonists,
pubmed-meshheading:20203300-Inflammation,
pubmed-meshheading:20203300-Inflammation Mediators,
pubmed-meshheading:20203300-Lipoproteins, LDL,
pubmed-meshheading:20203300-Lymphocytes,
pubmed-meshheading:20203300-Macrophages,
pubmed-meshheading:20203300-Male,
pubmed-meshheading:20203300-Mice,
pubmed-meshheading:20203300-Mice, Inbred C57BL,
pubmed-meshheading:20203300-Mice, Knockout,
pubmed-meshheading:20203300-Muscle, Smooth, Vascular,
pubmed-meshheading:20203300-Receptors, Histamine H1,
pubmed-meshheading:20203300-Receptors, Histamine H2
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pubmed:year |
2010
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pubmed:articleTitle |
Histamine H1 receptor promotes atherosclerotic lesion formation by increasing vascular permeability for low-density lipoproteins.
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pubmed:affiliation |
Cardiovascular Research, Institute of Physiology, University of Zurich, Switzerland.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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