| pubmed-article:20185813 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:20185813 | lifeskim:mentions | umls-concept:C0039005 | lld:lifeskim |
| pubmed-article:20185813 | lifeskim:mentions | umls-concept:C0003069 | lld:lifeskim |
| pubmed-article:20185813 | lifeskim:mentions | umls-concept:C0030274 | lld:lifeskim |
| pubmed-article:20185813 | lifeskim:mentions | umls-concept:C0271650 | lld:lifeskim |
| pubmed-article:20185813 | lifeskim:mentions | umls-concept:C0542341 | lld:lifeskim |
| pubmed-article:20185813 | lifeskim:mentions | umls-concept:C0221099 | lld:lifeskim |
| pubmed-article:20185813 | lifeskim:mentions | umls-concept:C1415067 | lld:lifeskim |
| pubmed-article:20185813 | lifeskim:mentions | umls-concept:C0392756 | lld:lifeskim |
| pubmed-article:20185813 | lifeskim:mentions | umls-concept:C1514485 | lld:lifeskim |
| pubmed-article:20185813 | pubmed:issue | 5 | lld:pubmed |
| pubmed-article:20185813 | pubmed:dateCreated | 2010-4-29 | lld:pubmed |
| pubmed-article:20185813 | pubmed:abstractText | The insulinotropic action of the incretin glucose-dependent insulinotropic polypeptide (GIP) is impaired in type 2 diabetes, while the effect of glucagon-like peptide-1 (GLP-1) is preserved. To evaluate the role of impaired GIP function in glucose homeostasis and development of the endocrine pancreas in a large animal model, we generated transgenic pigs expressing a dominant-negative GIP receptor (GIPR(dn)) in pancreatic islets. | lld:pubmed |
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| pubmed-article:20185813 | pubmed:language | eng | lld:pubmed |
| pubmed-article:20185813 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20185813 | pubmed:citationSubset | AIM | lld:pubmed |
| pubmed-article:20185813 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:20185813 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:20185813 | pubmed:month | May | lld:pubmed |
| pubmed-article:20185813 | pubmed:issn | 1939-327X | lld:pubmed |
| pubmed-article:20185813 | pubmed:author | pubmed-author:WolfEckhardE | lld:pubmed |
| pubmed-article:20185813 | pubmed:author | pubmed-author:HofmannAndrea... | lld:pubmed |
| pubmed-article:20185813 | pubmed:author | pubmed-author:PfeiferAlexan... | lld:pubmed |
| pubmed-article:20185813 | pubmed:author | pubmed-author:GökeBurkhardB | lld:pubmed |
| pubmed-article:20185813 | pubmed:author | pubmed-author:UlrichsKarinK | lld:pubmed |
| pubmed-article:20185813 | pubmed:author | pubmed-author:WankeRüdigerR | lld:pubmed |
| pubmed-article:20185813 | pubmed:author | pubmed-author:AmselgruberWe... | lld:pubmed |
| pubmed-article:20185813 | pubmed:author | pubmed-author:KesslerBarbar... | lld:pubmed |
| pubmed-article:20185813 | pubmed:author | pubmed-author:HerbachNadjaN | lld:pubmed |
| pubmed-article:20185813 | pubmed:author | pubmed-author:ChodnevskajaI... | lld:pubmed |
| pubmed-article:20185813 | pubmed:author | pubmed-author:MoskalenkoVas... | lld:pubmed |
| pubmed-article:20185813 | pubmed:author | pubmed-author:RennerSimoneS | lld:pubmed |
| pubmed-article:20185813 | pubmed:author | pubmed-author:von... | lld:pubmed |
| pubmed-article:20185813 | pubmed:author | pubmed-author:FehlingsChris... | lld:pubmed |
| pubmed-article:20185813 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:20185813 | pubmed:volume | 59 | lld:pubmed |
| pubmed-article:20185813 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:20185813 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:20185813 | pubmed:pagination | 1228-38 | lld:pubmed |
| pubmed-article:20185813 | pubmed:dateRevised | 2011-11-17 | lld:pubmed |
| pubmed-article:20185813 | pubmed:meshHeading | pubmed-meshheading:20185813... | lld:pubmed |
| pubmed-article:20185813 | pubmed:meshHeading | pubmed-meshheading:20185813... | lld:pubmed |
| pubmed-article:20185813 | pubmed:meshHeading | pubmed-meshheading:20185813... | lld:pubmed |
| pubmed-article:20185813 | pubmed:meshHeading | pubmed-meshheading:20185813... | lld:pubmed |
| pubmed-article:20185813 | pubmed:meshHeading | pubmed-meshheading:20185813... | lld:pubmed |
| pubmed-article:20185813 | pubmed:meshHeading | pubmed-meshheading:20185813... | lld:pubmed |
| pubmed-article:20185813 | pubmed:meshHeading | pubmed-meshheading:20185813... | lld:pubmed |
| pubmed-article:20185813 | pubmed:meshHeading | pubmed-meshheading:20185813... | lld:pubmed |
| pubmed-article:20185813 | pubmed:meshHeading | pubmed-meshheading:20185813... | lld:pubmed |
| pubmed-article:20185813 | pubmed:meshHeading | pubmed-meshheading:20185813... | lld:pubmed |
| pubmed-article:20185813 | pubmed:meshHeading | pubmed-meshheading:20185813... | lld:pubmed |
| pubmed-article:20185813 | pubmed:year | 2010 | lld:pubmed |
| pubmed-article:20185813 | pubmed:articleTitle | Glucose intolerance and reduced proliferation of pancreatic beta-cells in transgenic pigs with impaired glucose-dependent insulinotropic polypeptide function. | lld:pubmed |
| pubmed-article:20185813 | pubmed:affiliation | Chair for Molecular Animal Breeding and Biotechnology and Laboratory for Functional Genome Analysis, Gene Center, Ludwig Maximilians University (LMU) Munich, Munich, Germany. | lld:pubmed |
| pubmed-article:20185813 | pubmed:publicationType | Journal Article | lld:pubmed |
