20181814

Source:http://linkedlifedata.com/resource/pubmed/id/20181814

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Predicate	Object
rdf:type	pubmed:Citation
lifeskim:mentions	umls-concept:C0008976, umls-concept:C0009237, umls-concept:C0009830, umls-concept:C0011860, umls-concept:C0021368, umls-concept:C0035648, umls-concept:C1280500
pubmed:issue	4
pubmed:dateCreated	2010-3-22
pubmed:abstractText	Coffee consumption is associated with a decreased risk of type 2 diabetes. Suggested mechanisms underlying the association have included attenuation of subclinical inflammation and a reduction in oxidative stress.
pubmed:language	eng
pubmed:journal	http://linkedlifedata.com/resource/pubmed/journal/0376027
pubmed:citationSubset	AIM
pubmed:chemical	http://linkedlifedata.com/resource/pubmed/chemical/8-epi-prostaglandin F2alpha, http://linkedlifedata.com/resource/pubmed/chemical/Adiponectin, http://linkedlifedata.com/resource/pubmed/chemical/Anti-Inflammatory Agents, http://linkedlifedata.com/resource/pubmed/chemical/Apolipoproteins, http://linkedlifedata.com/resource/pubmed/chemical/Biological Markers, http://linkedlifedata.com/resource/pubmed/chemical/Blood Glucose, http://linkedlifedata.com/resource/pubmed/chemical/Caffeic Acids, http://linkedlifedata.com/resource/pubmed/chemical/Caffeine, http://linkedlifedata.com/resource/pubmed/chemical/Chlorogenic Acid, http://linkedlifedata.com/resource/pubmed/chemical/Cholesterol, http://linkedlifedata.com/resource/pubmed/chemical/Coffee, http://linkedlifedata.com/resource/pubmed/chemical/Dinoprost, http://linkedlifedata.com/resource/pubmed/chemical/Interleukin-18, http://linkedlifedata.com/resource/pubmed/chemical/Lipids, http://linkedlifedata.com/resource/pubmed/chemical/Plant Preparations, http://linkedlifedata.com/resource/pubmed/chemical/caffeic acid
pubmed:status	MEDLINE
pubmed:month	Apr
pubmed:issn	1938-3207
pubmed:author	pubmed-author:BidelSiamakS, pubmed-author:CarstensenMarenM, pubmed-author:ErlundIrisI, pubmed-author:HerderChristianC, pubmed-author:KempfKerstinK, pubmed-author:KoenigWolfgangW, pubmed-author:KolbHubertH, pubmed-author:KuhaSuviS, pubmed-author:MartinStephanS, pubmed-author:SundvallJoukoJ, pubmed-author:TuomilehtoJaakkoJ
pubmed:issnType	Electronic
pubmed:volume	91
pubmed:owner	NLM
pubmed:authorsComplete	Y
pubmed:pagination	950-7
pubmed:dateRevised	2010-9-27
pubmed:meshHeading	pubmed-meshheading:20181814-Adiponectin, pubmed-meshheading:20181814-Adult, pubmed-meshheading:20181814-Anti-Inflammatory Agents, pubmed-meshheading:20181814-Apolipoproteins, pubmed-meshheading:20181814-Biological Markers, pubmed-meshheading:20181814-Blood Glucose, pubmed-meshheading:20181814-Caffeic Acids, pubmed-meshheading:20181814-Caffeine, pubmed-meshheading:20181814-Chlorogenic Acid, pubmed-meshheading:20181814-Cholesterol, pubmed-meshheading:20181814-Coffee, pubmed-meshheading:20181814-Diabetes Mellitus, Type 2, pubmed-meshheading:20181814-Dinoprost, pubmed-meshheading:20181814-Female, pubmed-meshheading:20181814-Humans, pubmed-meshheading:20181814-Inflammation, pubmed-meshheading:20181814-Interleukin-18, pubmed-meshheading:20181814-Lipids, pubmed-meshheading:20181814-Male, pubmed-meshheading:20181814-Middle Aged, pubmed-meshheading:20181814-Plant Preparations, pubmed-meshheading:20181814-Risk Factors, pubmed-meshheading:20181814-Single-Blind Method
pubmed:year	2010
pubmed:articleTitle	Effects of coffee consumption on subclinical inflammation and other risk factors for type 2 diabetes: a clinical trial.
pubmed:affiliation	Institute of Clinical Diabetology, German Diabetes Center, Leibniz Center for Diabetes Research at Heinrich, Heine University Düsseldorf, Düsseldorf, Germany.
pubmed:publicationType	Journal Article, Research Support, Non-U.S. Gov't, Clinical Trial, Phase III