Linked Life Data

20176206

Source:http://linkedlifedata.com/resource/pubmed/id/20176206

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
3
pubmed:dateCreated
2010-2-23
pubmed:abstractText
Esophageal adenocarcinoma is thought to arise from lesions produced by chronic esophageal inflammation. Secretory phospholipase A(2) is an important mediator of mucosal response to gastroesophageal reflux, but its role in the function of mature cancer cells is unclear. We sought to determine the influence of group IIa secretory phospholipase A(2) on proliferation of human esophageal adenocarcinoma cells.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
AIM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Mar
pubmed:issn
1097-685X
pubmed:author
pubmed:copyrightInfo
Copyright 2010 The American Association for Thoracic Surgery. Published by Mosby, Inc. All rights reserved.
pubmed:issnType
Electronic
pubmed:volume
139
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
591-9; discussion 599
pubmed:meshHeading
pubmed:year
2010
pubmed:articleTitle
Modulation of growth in human esophageal adenocarcinoma cells by group IIa secretory phospholipase A(2).
pubmed:affiliation
Department of Surgery, Division of Cardiothoracic Surgery, University of Colorado Denver School of Medicine, Aurora, CO, USA.
pubmed:publicationType
Journal Article, Research Support, Non-U.S. Gov't