20167660

Source:http://linkedlifedata.com/resource/pubmed/id/20167660

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Predicate	Object
rdf:type	pubmed:Citation
lifeskim:mentions	umls-concept:C0014257, umls-concept:C0021368, umls-concept:C0023779, umls-concept:C0027996, umls-concept:C0032105, umls-concept:C0205178, umls-concept:C0277785, umls-concept:C0332120, umls-concept:C0332161, umls-concept:C0332291, umls-concept:C0392747, umls-concept:C0443172, umls-concept:C1801960
pubmed:issue	5
pubmed:dateCreated	2010-4-15
pubmed:abstractText	To determine if niacin can confer cardiovascular benefit by inhibiting vascular inflammation and improving endothelial function independent of changes in plasma lipid and lipoprotein levels.
pubmed:language	eng
pubmed:journal	http://linkedlifedata.com/resource/pubmed/journal/9505803
pubmed:citationSubset	IM
pubmed:chemical	http://linkedlifedata.com/resource/pubmed/chemical/Anti-Inflammatory Agents, http://linkedlifedata.com/resource/pubmed/chemical/Chemokine CCL2, http://linkedlifedata.com/resource/pubmed/chemical/Cyclic GMP, http://linkedlifedata.com/resource/pubmed/chemical/Free Radical Scavengers, http://linkedlifedata.com/resource/pubmed/chemical/Glutathione, http://linkedlifedata.com/resource/pubmed/chemical/Intercellular Adhesion Molecule-1, http://linkedlifedata.com/resource/pubmed/chemical/Lipids, http://linkedlifedata.com/resource/pubmed/chemical/Niacin, http://linkedlifedata.com/resource/pubmed/chemical/Peroxidase, http://linkedlifedata.com/resource/pubmed/chemical/Reactive Oxygen Species, http://linkedlifedata.com/resource/pubmed/chemical/Tumor Necrosis Factor-alpha, http://linkedlifedata.com/resource/pubmed/chemical/Vascular Cell Adhesion Molecule-1, http://linkedlifedata.com/resource/pubmed/chemical/Vasodilator Agents
pubmed:status	MEDLINE
pubmed:month	May
pubmed:issn	1524-4636
pubmed:author	pubmed-author:BarterPhilip JPJ, pubmed-author:CharltonFrancescaF, pubmed-author:MaesE MEM, pubmed-author:RyeKerry-AnneKA, pubmed-author:WittingPaulP, pubmed-author:WuBen JBJ
pubmed:issnType	Electronic
pubmed:volume	30
pubmed:owner	NLM
pubmed:authorsComplete	Y
pubmed:pagination	968-75
pubmed:meshHeading	pubmed-meshheading:20167660-Animals, pubmed-meshheading:20167660-Anti-Inflammatory Agents, pubmed-meshheading:20167660-Aortic Diseases, pubmed-meshheading:20167660-Carotid Artery Diseases, pubmed-meshheading:20167660-Chemokine CCL2, pubmed-meshheading:20167660-Cyclic GMP, pubmed-meshheading:20167660-Disease Models, Animal, pubmed-meshheading:20167660-Dose-Response Relationship, Drug, pubmed-meshheading:20167660-Endothelium, Vascular, pubmed-meshheading:20167660-Free Radical Scavengers, pubmed-meshheading:20167660-Glutathione, pubmed-meshheading:20167660-Inflammation, pubmed-meshheading:20167660-Intercellular Adhesion Molecule-1, pubmed-meshheading:20167660-Lipids, pubmed-meshheading:20167660-Neutrophil Infiltration, pubmed-meshheading:20167660-Niacin, pubmed-meshheading:20167660-Oxidation-Reduction, pubmed-meshheading:20167660-Peroxidase, pubmed-meshheading:20167660-Rabbits, pubmed-meshheading:20167660-Reactive Oxygen Species, pubmed-meshheading:20167660-Tumor Necrosis Factor-alpha, pubmed-meshheading:20167660-Vascular Cell Adhesion Molecule-1, pubmed-meshheading:20167660-Vasodilation, pubmed-meshheading:20167660-Vasodilator Agents
pubmed:year	2010
pubmed:articleTitle	Evidence that niacin inhibits acute vascular inflammation and improves endothelial dysfunction independent of changes in plasma lipids.
pubmed:affiliation	Lipid Research Group, Heart Research Institute, 7 Eliza St, Newtown, New South Wales, Australia 2042.
pubmed:publicationType	Journal Article, Research Support, Non-U.S. Gov't