pubmed-article:20154604 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:20154604 | lifeskim:mentions | umls-concept:C0007587 | lld:lifeskim |
pubmed-article:20154604 | lifeskim:mentions | umls-concept:C0242488 | lld:lifeskim |
pubmed-article:20154604 | lifeskim:mentions | umls-concept:C0439852 | lld:lifeskim |
pubmed-article:20154604 | lifeskim:mentions | umls-concept:C0814423 | lld:lifeskim |
pubmed-article:20154604 | lifeskim:mentions | umls-concept:C0302350 | lld:lifeskim |
pubmed-article:20154604 | lifeskim:mentions | umls-concept:C1554080 | lld:lifeskim |
pubmed-article:20154604 | lifeskim:mentions | umls-concept:C1706198 | lld:lifeskim |
pubmed-article:20154604 | pubmed:issue | 4 | lld:pubmed |
pubmed-article:20154604 | pubmed:dateCreated | 2010-3-25 | lld:pubmed |
pubmed-article:20154604 | pubmed:abstractText | Indirect acute lung injury is associated with high morbidity and mortality. However, the underlying pathophysiology is only marginally understood, and so far no pathophysiologic-based remedy exists. We hypothesized that apoptosis of lung epithelial cells is a pathophysiological relevant process in the development of indirect acute lung injury and that it should be accessible to a siRNA-based therapeutic intervention in vivo. | lld:pubmed |
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pubmed-article:20154604 | pubmed:language | eng | lld:pubmed |
pubmed-article:20154604 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:20154604 | pubmed:citationSubset | AIM | lld:pubmed |
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pubmed-article:20154604 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:20154604 | pubmed:month | Apr | lld:pubmed |
pubmed-article:20154604 | pubmed:issn | 1530-0293 | lld:pubmed |
pubmed-article:20154604 | pubmed:author | pubmed-author:AyalaAlfredA | lld:pubmed |
pubmed-article:20154604 | pubmed:author | pubmed-author:ChungChun-Shi... | lld:pubmed |
pubmed-article:20154604 | pubmed:author | pubmed-author:PerlMarioM | lld:pubmed |
pubmed-article:20154604 | pubmed:author | pubmed-author:Lomas-NeiraJo... | lld:pubmed |
pubmed-article:20154604 | pubmed:author | pubmed-author:PerlUlrikeU | lld:pubmed |
pubmed-article:20154604 | pubmed:author | pubmed-author:ThakkarRajanR | lld:pubmed |
pubmed-article:20154604 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:20154604 | pubmed:volume | 38 | lld:pubmed |
pubmed-article:20154604 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:20154604 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:20154604 | pubmed:pagination | 1179-86 | lld:pubmed |
pubmed-article:20154604 | pubmed:dateRevised | 2010-12-3 | lld:pubmed |
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pubmed-article:20154604 | pubmed:year | 2010 | lld:pubmed |
pubmed-article:20154604 | pubmed:articleTitle | Therapeutic accessibility of caspase-mediated cell death as a key pathomechanism in indirect acute lung injury. | lld:pubmed |
pubmed-article:20154604 | pubmed:affiliation | Shock-Trauma Research Laboratories in the Division of Surgical Research and Department of Surgery, Rhode Island Hospital and Brown University, School of Medicine, Providence, RI, USA. | lld:pubmed |
pubmed-article:20154604 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:20154604 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
pubmed-article:20154604 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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