Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
2
pubmed:dateCreated
2010-2-5
pubmed:abstractText
Clear cell adenocarcinoma of the ovary often shows resistance to anticancer agents. It accounts for 20% of epithelial ovarian cancer in Japan versus around 5% in other countries. We investigated new molecules to use when developing molecular-targeting therapy for clear cell adenocarcinoma.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Feb
pubmed:issn
1525-1438
pubmed:author
pubmed:issnType
Electronic
pubmed:volume
20
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
212-9
pubmed:dateRevised
2011-11-17
pubmed:meshHeading
pubmed-meshheading:20134265-Adenocarcinoma, Clear Cell, pubmed-meshheading:20134265-Animals, pubmed-meshheading:20134265-Cell Line, Tumor, pubmed-meshheading:20134265-Cell Proliferation, pubmed-meshheading:20134265-Female, pubmed-meshheading:20134265-Humans, pubmed-meshheading:20134265-Male, pubmed-meshheading:20134265-Mice, pubmed-meshheading:20134265-Mice, Inbred BALB C, pubmed-meshheading:20134265-Mice, Nude, pubmed-meshheading:20134265-Multienzyme Complexes, pubmed-meshheading:20134265-Oligonucleotide Array Sequence Analysis, pubmed-meshheading:20134265-Ovarian Neoplasms, pubmed-meshheading:20134265-POU Domain Factors, pubmed-meshheading:20134265-Phosphodiesterase I, pubmed-meshheading:20134265-Phosphoric Diester Hydrolases, pubmed-meshheading:20134265-Pyrophosphatases, pubmed-meshheading:20134265-RNA, Small Interfering, pubmed-meshheading:20134265-Receptors, Lysophosphatidic Acid
pubmed:year
2010
pubmed:articleTitle
Transcription factor POU6F1 is important for proliferation of clear cell adenocarcinoma of the ovary and is a potential new molecular target.
pubmed:affiliation
Department of Obstetrics and Gynecology, St Marianna University School of Medicine, Kanagawa, Japan. nao@marianna-u.ac.jp
pubmed:publicationType
Journal Article, Research Support, Non-U.S. Gov't