rdf:type |
|
lifeskim:mentions |
umls-concept:C0027950,
umls-concept:C0030685,
umls-concept:C0039194,
umls-concept:C0085295,
umls-concept:C0085297,
umls-concept:C0085358,
umls-concept:C0205314,
umls-concept:C0220781,
umls-concept:C0391871,
umls-concept:C0441712,
umls-concept:C0574032,
umls-concept:C0679622,
umls-concept:C0680255,
umls-concept:C1283071,
umls-concept:C1332717,
umls-concept:C1413244,
umls-concept:C1533691,
umls-concept:C1706438,
umls-concept:C1709059,
umls-concept:C1883254,
umls-concept:C1963578,
umls-concept:C2348480,
umls-concept:C2349974,
umls-concept:C2698600
|
pubmed:issue |
3
|
pubmed:dateCreated |
2010-5-21
|
pubmed:abstractText |
Numerous mechanisms have been proposed to explain the beneficial action of intravenous immune globulin (IVIG) in autoimmune and systemic inflammatory disorders; among others, they could decrease pro-inflammatory cytokine levels and also induce anti-inflammatory cytokines.
|
pubmed:language |
eng
|
pubmed:journal |
|
pubmed:citationSubset |
IM
|
pubmed:chemical |
|
pubmed:status |
MEDLINE
|
pubmed:month |
May
|
pubmed:issn |
1573-2592
|
pubmed:author |
|
pubmed:issnType |
Electronic
|
pubmed:volume |
30
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
384-92
|
pubmed:meshHeading |
pubmed-meshheading:20127276-CD8-Positive T-Lymphocytes,
pubmed-meshheading:20127276-Cell Separation,
pubmed-meshheading:20127276-Cells, Cultured,
pubmed-meshheading:20127276-Drug Contamination,
pubmed-meshheading:20127276-Fas Ligand Protein,
pubmed-meshheading:20127276-Fatigue Syndrome, Chronic,
pubmed-meshheading:20127276-Flow Cytometry,
pubmed-meshheading:20127276-HIV,
pubmed-meshheading:20127276-HIV Infections,
pubmed-meshheading:20127276-Histocompatibility Antigens Class I,
pubmed-meshheading:20127276-Humans,
pubmed-meshheading:20127276-Immunoglobulins, Intravenous,
pubmed-meshheading:20127276-Immunologic Factors,
pubmed-meshheading:20127276-Immunotherapy,
pubmed-meshheading:20127276-Interleukin-10,
pubmed-meshheading:20127276-Neutrophils,
pubmed-meshheading:20127276-Systemic Vasculitis,
pubmed-meshheading:20127276-Transforming Growth Factor beta1
|
pubmed:year |
2010
|
pubmed:articleTitle |
sHLA-I Contamination, a novel mechanism to explain ex vivo/in vitro modulation of IL-10 synthesis and release in CD8(+) T lymphocytes and in neutrophils following intravenous immunoglobulin infusion.
|
pubmed:affiliation |
Division of Clinical Immunology, Department of Internal Medicine, University of Genoa Medical School, 16132, Genoa, Italy. mghio@unige.it
|
pubmed:publicationType |
Journal Article
|