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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
1
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pubmed:dateCreated |
2010-1-29
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pubmed:abstractText |
Perturbations in the gut microbiota have been linked to atopic diseases. However, the development of atopic diseases depends not only on environmental factors (like microbial stimulation) but also on genetic factors. It is likely that particularly gene-environmental interactions in early life determine the development of atopy.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
AIM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Jan
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pubmed:issn |
1097-6825
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pubmed:author |
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pubmed:copyrightInfo |
Copyright 2010 American Academy of Allergy, Asthma & Immunology. Published by Mosby, Inc. All rights reserved.
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pubmed:issnType |
Electronic
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pubmed:volume |
125
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
231-6.e1-5
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pubmed:meshHeading |
pubmed-meshheading:20109750-Antigens, CD14,
pubmed-meshheading:20109750-Child, Preschool,
pubmed-meshheading:20109750-Dermatitis, Atopic,
pubmed-meshheading:20109750-Escherichia coli,
pubmed-meshheading:20109750-Feces,
pubmed-meshheading:20109750-Genetic Predisposition to Disease,
pubmed-meshheading:20109750-Humans,
pubmed-meshheading:20109750-Hypersensitivity, Immediate,
pubmed-meshheading:20109750-Infant,
pubmed-meshheading:20109750-Infant, Newborn,
pubmed-meshheading:20109750-Polymorphism, Single Nucleotide,
pubmed-meshheading:20109750-Toll-Like Receptor 4
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pubmed:year |
2010
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pubmed:articleTitle |
Host-microbial interactions in childhood atopy: toll-like receptor 4 (TLR4), CD14, and fecal Escherichia coli.
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pubmed:affiliation |
Department of Epidemiology, Nutrition and Toxicology Research Institute Maastricht and Care and Public Health Research Institute, Maastricht University, PO Box 616, 6200 MD Maastricht, The Netherlands. j.penders@epid.unimaas.nl
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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