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20103675
Source:
http://linkedlifedata.com/resource/pubmed/id/20103675
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51
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Statements in which the resource exists as a subject.
Predicate
Object
rdf:type
pubmed:Citation
lifeskim:mentions
umls-concept:C0007634
,
umls-concept:C0037083
,
umls-concept:C0164786
,
umls-concept:C0232901
,
umls-concept:C0308269
,
umls-concept:C0812228
,
umls-concept:C1514559
,
umls-concept:C1710082
,
umls-concept:C1999177
,
umls-concept:C2239176
pubmed:issue
3
pubmed:dateCreated
2010-2-3
pubmed:abstractText
This study aims to profile the expressions of 156 microRNAs (miRNA) in hepatocellular carcinoma (HCC) and to characterize the functions of miR-222, the most significantly upregulated candidate identified.
pubmed:language
eng
pubmed:journal
http://linkedlifedata.com/resource/pubmed/journal/9502500
pubmed:citationSubset
IM
pubmed:chemical
http://linkedlifedata.com/resource/pubmed/chemical/MIRN222 microRNA, human
,
http://linkedlifedata.com/resource/pubmed/chemical/MicroRNAs
,
http://linkedlifedata.com/resource/pubmed/chemical/Proto-Oncogene Proteins c-akt
pubmed:status
MEDLINE
pubmed:month
Feb
pubmed:issn
1078-0432
pubmed:author
pubmed-author:ChanAnthony W-HAW
,
pubmed-author:ChoyKwong-WaiKW
,
pubmed-author:LaiPaul B-SPB
,
pubmed-author:OnerciTevfik MetinTM
,
pubmed-author:ToKa-FaiKF
,
pubmed-author:WongNathalieN
,
pubmed-author:WongQueenie W-LQW
pubmed:issnType
Print
pubmed:day
1
pubmed:volume
16
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
867-75
pubmed:meshHeading
pubmed-meshheading:20103675-Carcinoma, Hepatocellular
,
pubmed-meshheading:20103675-Cell Movement
,
pubmed-meshheading:20103675-Disease Progression
,
pubmed-meshheading:20103675-Humans
,
pubmed-meshheading:20103675-Liver Cirrhosis
,
pubmed-meshheading:20103675-Liver Neoplasms
,
pubmed-meshheading:20103675-MicroRNAs
,
pubmed-meshheading:20103675-Proto-Oncogene Proteins c-akt
,
pubmed-meshheading:20103675-Signal Transduction
,
pubmed-meshheading:20103675-Up-Regulation
pubmed:year
2010
pubmed:articleTitle
MiR-222 overexpression confers cell migratory advantages in hepatocellular carcinoma through enhancing AKT signaling.
pubmed:affiliation
Department of Anatomical and Cellular Pathology, The Chinese University of Hong Kong, Shatin, Hong Kong, China.
pubmed:publicationType
Journal Article
,
Research Support, Non-U.S. Gov't