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pubmed-article:20101459pubmed:abstractTextCancer cells are thought to possess immune evasion properties due to FasL overexpression in many types of human tumors. In the present study, we set out to investigate the role of MAPK-ERK pathway in 67-kDa laminin receptor induced FasL expression and FasL-mediated apoptosis in human cholangiocarcinoma cells.lld:pubmed
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pubmed-article:20101459pubmed:articleTitleThe role of MAPK-ERK pathway in 67-kDa laminin receptor-induced FasL expression in human cholangiocarcinoma cells.lld:pubmed
pubmed-article:20101459pubmed:affiliationHepatobiliary Surgery Hospital and Institute, Southwest Hospital, Third Military Medical University, 400038, Chongqing, China.lld:pubmed
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