Linked Life Data

20088932

Source:http://linkedlifedata.com/resource/pubmed/id/20088932

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
4
pubmed:dateCreated
2010-4-19
pubmed:abstractText
The fibrinolytic system has been implicated in the pathogenesis of pulmonary hypertension (PH). Thrombin-activatable fibrinolysis inhibitor (TAFI) inhibits fibrinolysis and therefore its absence would be expected to increase fibrinolysis and ameliorate PH.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Apr
pubmed:issn
1538-7836
pubmed:author
pubmed:issnType
Electronic
pubmed:volume
8
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
808-16
pubmed:meshHeading
pubmed-meshheading:20088932-Animals, pubmed-meshheading:20088932-Biological Markers, pubmed-meshheading:20088932-Bronchoalveolar Lavage Fluid, pubmed-meshheading:20088932-Capillary Permeability, pubmed-meshheading:20088932-Carboxypeptidase U, pubmed-meshheading:20088932-Chemokine CCL2, pubmed-meshheading:20088932-Disease Models, Animal, pubmed-meshheading:20088932-Fibrinolysis, pubmed-meshheading:20088932-Hypertension, Pulmonary, pubmed-meshheading:20088932-Hypertrophy, Right Ventricular, pubmed-meshheading:20088932-Inflammation Mediators, pubmed-meshheading:20088932-Interleukin-6, pubmed-meshheading:20088932-Lung, pubmed-meshheading:20088932-Male, pubmed-meshheading:20088932-Mice, pubmed-meshheading:20088932-Mice, Inbred C57BL, pubmed-meshheading:20088932-Mice, Knockout, pubmed-meshheading:20088932-Monocrotaline, pubmed-meshheading:20088932-Platelet-Derived Growth Factor, pubmed-meshheading:20088932-Pulmonary Artery, pubmed-meshheading:20088932-Transforming Growth Factor beta1, pubmed-meshheading:20088932-Tumor Necrosis Factor-alpha, pubmed-meshheading:20088932-Weight Loss
pubmed:year
2010
pubmed:articleTitle
Pulmonary hypertension is ameliorated in mice deficient in thrombin-activatable fibrinolysis inhibitor.
pubmed:affiliation
Department of Immunology, Mie University School of Medicine, Edobashi 2-174, Tsu city, Mie 514-8507, Japan.
pubmed:publicationType
Journal Article, Research Support, Non-U.S. Gov't