Source:http://linkedlifedata.com/resource/pubmed/id/20087176
Switch to
Predicate | Object |
---|---|
rdf:type | |
lifeskim:mentions | |
pubmed:issue |
2
|
pubmed:dateCreated |
2010-2-10
|
pubmed:abstractText |
The pathophysiology of Philadelphia-chromosome negative myeloproliferative disorders has significantly advanced with the discovery of JAK2V617F. The prevalence of JAK2V617F mutation has made it a much anticipated target for inhibition; this review will update and assess progress.
|
pubmed:language |
eng
|
pubmed:journal | |
pubmed:citationSubset |
IM
|
pubmed:chemical | |
pubmed:status |
MEDLINE
|
pubmed:month |
Mar
|
pubmed:issn |
1531-7048
|
pubmed:author | |
pubmed:issnType |
Electronic
|
pubmed:volume |
17
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
110-6
|
pubmed:meshHeading | |
pubmed:year |
2010
|
pubmed:articleTitle |
JAK2 kinase inhibitors and myeloproliferative disorders.
|
pubmed:affiliation |
Hematology and Medical Oncology, University of Utah, Salt Lake City, Utah, USA.
|
pubmed:publicationType |
Journal Article,
Review
|