| pubmed-article:20042177 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:20042177 | lifeskim:mentions | umls-concept:C0242383 | lld:lifeskim |
| pubmed-article:20042177 | lifeskim:mentions | umls-concept:C0017337 | lld:lifeskim |
| pubmed-article:20042177 | lifeskim:mentions | umls-concept:C1280500 | lld:lifeskim |
| pubmed-article:20042177 | lifeskim:mentions | umls-concept:C0205419 | lld:lifeskim |
| pubmed-article:20042177 | lifeskim:mentions | umls-concept:C1121571 | lld:lifeskim |
| pubmed-article:20042177 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:20042177 | pubmed:dateCreated | 2010-2-22 | lld:pubmed |
| pubmed-article:20042177 | pubmed:abstractText | Age-related macular degeneration (AMD) is the leading cause of blindness among older adults, in which oxidative damage may play a pivotal role. Paraoxonase 1 (PON1) protects against oxidative damage and has been evaluated for its involvement in aging diseases including AMD. This study investigated whether PON1 gene polymorphisms associate with AMD. | lld:pubmed |
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| pubmed-article:20042177 | pubmed:language | eng | lld:pubmed |
| pubmed-article:20042177 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:20042177 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:20042177 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:20042177 | pubmed:month | Mar | lld:pubmed |
| pubmed-article:20042177 | pubmed:issn | 1879-1891 | lld:pubmed |
| pubmed-article:20042177 | pubmed:author | pubmed-author:PeacheyNeal... | lld:pubmed |
| pubmed-article:20042177 | pubmed:author | pubmed-author:SturgillGwen... | lld:pubmed |
| pubmed-article:20042177 | pubmed:author | pubmed-author:HagstromSteph... | lld:pubmed |
| pubmed-article:20042177 | pubmed:author | pubmed-author:PauerGayle... | lld:pubmed |
| pubmed-article:20042177 | pubmed:author | pubmed-author:Clinical... | lld:pubmed |
| pubmed-article:20042177 | pubmed:copyrightInfo | (c) 2010 Elsevier Inc. All rights reserved. | lld:pubmed |
| pubmed-article:20042177 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:20042177 | pubmed:volume | 149 | lld:pubmed |
| pubmed-article:20042177 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:20042177 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:20042177 | pubmed:pagination | 513-22 | lld:pubmed |
| pubmed-article:20042177 | pubmed:dateRevised | 2011-7-20 | lld:pubmed |
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| pubmed-article:20042177 | pubmed:year | 2010 | lld:pubmed |
| pubmed-article:20042177 | pubmed:articleTitle | Protective effect of paraoxonase 1 gene variant Gln192Arg in age-related macular degeneration. | lld:pubmed |
| pubmed-article:20042177 | pubmed:affiliation | Department of Ophthalmic Research, Cole Eye Institute, Cleveland Clinic, Cleveland, Ohio 44195, USA. | lld:pubmed |
| pubmed-article:20042177 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:20042177 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
| pubmed-article:20042177 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| pubmed-article:20042177 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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