Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
1
pubmed:dateCreated
2009-12-30
pubmed:abstractText
Interaction of receptor for advanced glycation end products (RAGE) with advanced glycation end products (AGEs) is an important pathogenic mechanism of diabetic complications. Three mutations in the promoter region of the RAGE gene (T-429C, T-374A and a 63 bp deletion spanning from -407 to -345 nucleotides) were known to have increased transcriptional activities. We investigated the relationship between these polymorphisms and the risk of cardiovascular diseases in Chinese subjects with overt diabetic nephropathy.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Jan
pubmed:issn
0301-0430
pubmed:author
pubmed:issnType
Print
pubmed:volume
73
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
44-50
pubmed:dateRevised
2010-11-18
pubmed:meshHeading
pubmed-meshheading:20040351-Analysis of Variance, pubmed-meshheading:20040351-Asian Continental Ancestry Group, pubmed-meshheading:20040351-Cardiovascular Diseases, pubmed-meshheading:20040351-Chi-Square Distribution, pubmed-meshheading:20040351-China, pubmed-meshheading:20040351-Diabetic Nephropathies, pubmed-meshheading:20040351-Disease-Free Survival, pubmed-meshheading:20040351-Genetic Predisposition to Disease, pubmed-meshheading:20040351-Genotype, pubmed-meshheading:20040351-Humans, pubmed-meshheading:20040351-Kaplan-Meier Estimate, pubmed-meshheading:20040351-Kidney Failure, Chronic, pubmed-meshheading:20040351-Polymerase Chain Reaction, pubmed-meshheading:20040351-Polymorphism, Genetic, pubmed-meshheading:20040351-Proportional Hazards Models, pubmed-meshheading:20040351-Receptors, Immunologic, pubmed-meshheading:20040351-Risk Factors
pubmed:year
2010
pubmed:articleTitle
Relation between polymorphisms of receptor for advanced glycation end products (RAGE) and cardiovascular diseases in Chinese patients with diabetic nephropathy.
pubmed:affiliation
Department of Medicine and Therapeutics, Prince of Wales Hospital, The Chinese University of Hong Kong, Shatin, Hong Kong, China.
pubmed:publicationType
Journal Article, Research Support, Non-U.S. Gov't