Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
2
pubmed:dateCreated
2010-2-9
pubmed:abstractText
Smoking not only increases the risk that coronary heart disease will develop but also morbidity and mortality in patients with known coronary atherosclerosis and after coronary artery bypass grafting. Excessive generation of reactive oxygen species (ROS) has been implicated as the final common pathway for the development of endothelial dysfunction in various cardiovascular risk factors. This study assessed the influence of smoking on two different human arteries routinely used as coronary artery bypass graft conduits.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Feb
pubmed:issn
1097-6809
pubmed:author
pubmed:copyrightInfo
Copyright 2010 Society for Vascular Surgery. Published by Mosby, Inc. All rights reserved.
pubmed:issnType
Electronic
pubmed:volume
51
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
438-44
pubmed:meshHeading
pubmed-meshheading:20036100-Acetylcholine, pubmed-meshheading:20036100-Aged, pubmed-meshheading:20036100-Coronary Artery Bypass, pubmed-meshheading:20036100-Dose-Response Relationship, Drug, pubmed-meshheading:20036100-Endothelin-1, pubmed-meshheading:20036100-Female, pubmed-meshheading:20036100-Humans, pubmed-meshheading:20036100-Male, pubmed-meshheading:20036100-Mammary Arteries, pubmed-meshheading:20036100-Microscopy, Confocal, pubmed-meshheading:20036100-Middle Aged, pubmed-meshheading:20036100-Nitroprusside, pubmed-meshheading:20036100-Norepinephrine, pubmed-meshheading:20036100-Oxidative Stress, pubmed-meshheading:20036100-Radial Artery, pubmed-meshheading:20036100-Reactive Oxygen Species, pubmed-meshheading:20036100-Smoking, pubmed-meshheading:20036100-Vasoconstriction, pubmed-meshheading:20036100-Vasoconstrictor Agents, pubmed-meshheading:20036100-Vasodilation, pubmed-meshheading:20036100-Vasodilator Agents
pubmed:year
2010
pubmed:articleTitle
Reactive oxygen species mediate functional differences in human radial and internal thoracic arteries from smokers.
pubmed:affiliation
Heart Surgery Center Basel-Bern, University Hospital, Bern, Switzerland. dremueller@bluewin.ch
pubmed:publicationType
Journal Article, Comparative Study, Research Support, Non-U.S. Gov't