Source:http://linkedlifedata.com/resource/pubmed/id/19951577
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
21
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pubmed:dateCreated |
2009-12-2
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pubmed:abstractText |
IgA nephropathy is the major cause of end-stage renal failure in patients with primary glomerular diseases. Tumor suppressor cylindromatosis (CYLD), the recently identified member of the deubiquitinating enzymes, has been actively involved in regulation of inflammation. This study was undertaken to investigate the CYLD expression profile in IgA nephropathy and identify factors associated with CYLD expression.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
Nov
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pubmed:issn |
0366-6999
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:day |
5
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pubmed:volume |
122
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
2603-7
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pubmed:meshHeading |
pubmed-meshheading:19951577-Glomerular Filtration Rate,
pubmed-meshheading:19951577-Glomerulonephritis, IGA,
pubmed-meshheading:19951577-Humans,
pubmed-meshheading:19951577-Immunohistochemistry,
pubmed-meshheading:19951577-Kidney Diseases,
pubmed-meshheading:19951577-Logistic Models,
pubmed-meshheading:19951577-Proteinuria,
pubmed-meshheading:19951577-Tumor Suppressor Proteins
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pubmed:year |
2009
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pubmed:articleTitle |
Tumor suppressor cylindromatosis: expressed in IgA nephropathy and negatively associated with renal tubulo-interstitial lesion.
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pubmed:affiliation |
Department of Nephrology, Beijing Chaoyang Hospital, Capital Medical University, Beijing 100025, China.
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pubmed:publicationType |
Journal Article
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