pubmed-article:19884805 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:19884805 | lifeskim:mentions | umls-concept:C0567498 | lld:lifeskim |
pubmed-article:19884805 | lifeskim:mentions | umls-concept:C0677898 | lld:lifeskim |
pubmed-article:19884805 | lifeskim:mentions | umls-concept:C0085187 | lld:lifeskim |
pubmed-article:19884805 | lifeskim:mentions | umls-concept:C0221198 | lld:lifeskim |
pubmed-article:19884805 | lifeskim:mentions | umls-concept:C1444754 | lld:lifeskim |
pubmed-article:19884805 | lifeskim:mentions | umls-concept:C0012899 | lld:lifeskim |
pubmed-article:19884805 | pubmed:issue | 4 | lld:pubmed |
pubmed-article:19884805 | pubmed:dateCreated | 2010-8-6 | lld:pubmed |
pubmed-article:19884805 | pubmed:abstractText | Carcinogenesis is a multistep process involving the accumulation of genetic and molecular abnormalities. It has been suggested that there is a relationship between telomere attrition in the early stages of carcinogenesis and activation of the DNA damage response machinery. We explored telomere length modification and damage response pathway activation at 3 steps of breast carcinogenesis. | lld:pubmed |
pubmed-article:19884805 | pubmed:language | eng | lld:pubmed |
pubmed-article:19884805 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19884805 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:19884805 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19884805 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:19884805 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:19884805 | pubmed:month | Aug | lld:pubmed |
pubmed-article:19884805 | pubmed:issn | 1537-453X | lld:pubmed |
pubmed-article:19884805 | pubmed:author | pubmed-author:SoriaJean-Cha... | lld:pubmed |
pubmed-article:19884805 | pubmed:author | pubmed-author:SabatierLaure... | lld:pubmed |
pubmed-article:19884805 | pubmed:author | pubmed-author:MathieuMarie-... | lld:pubmed |
pubmed-article:19884805 | pubmed:author | pubmed-author:AndréFabriceF | lld:pubmed |
pubmed-article:19884805 | pubmed:author | pubmed-author:NuciforoPaolo... | lld:pubmed |
pubmed-article:19884805 | pubmed:author | pubmed-author:DelalogeSuzet... | lld:pubmed |
pubmed-article:19884805 | pubmed:author | pubmed-author:HernandezJuan... | lld:pubmed |
pubmed-article:19884805 | pubmed:author | pubmed-author:RaynaudChrist... | lld:pubmed |
pubmed-article:19884805 | pubmed:author | pubmed-author:CommoFrederic... | lld:pubmed |
pubmed-article:19884805 | pubmed:author | pubmed-author:LlorcaFrédéri... | lld:pubmed |
pubmed-article:19884805 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:19884805 | pubmed:volume | 33 | lld:pubmed |
pubmed-article:19884805 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:19884805 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:19884805 | pubmed:pagination | 341-5 | lld:pubmed |
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pubmed-article:19884805 | pubmed:meshHeading | pubmed-meshheading:19884805... | lld:pubmed |
pubmed-article:19884805 | pubmed:year | 2010 | lld:pubmed |
pubmed-article:19884805 | pubmed:articleTitle | DNA damage repair and telomere length in normal breast, preneoplastic lesions, and invasive cancer. | lld:pubmed |
pubmed-article:19884805 | pubmed:affiliation | Département des Science du Vinvant-Laboratoire de radiobiologie et oncologie, CEA, Fontenay-aux-Roses, France. | lld:pubmed |
pubmed-article:19884805 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:19884805 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |