Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
3
pubmed:dateCreated
2010-6-23
pubmed:abstractText
By using kerosene and avoiding postmortem rigor one can obtain perfusion rates in kidneys nearly five times faster than those reported by observers who perfused kidneys immediately post mortem with saline solution, only half as viscous as kerosene. The results obtained by kerosene perfusion indicate possible renal blood flow 50 to 100 per cent greater than that measured by Smith and his coworkers (7) in living men by diodrast clearance under normal conditions, and about as high as those observed in febrile subjects. Like the diodrast method, kerosene perfusion shows a striking decrease in renal vascular bed between early matuity (age 18 to 35) and senescence (45 to 60). This decrease is about 25 per cent. Most kidneys from patients with hypertension without uremia have vascular beds in the normal range, but a few show great decreases in capacity for blood flow. This evidence is interpreted as another indication that renal arteriosclerosis is often a result, rarely a cause of hypertension. Significant occlusion of large renal arteries is rare. Uremia due to amyloid may occur with no significant decrease in renal vascular bed, but the uremia of renal sclerosis, glomerulo- or pyelonephritis is associated with reduction of vascular bed to very low levels.
pubmed:commentsCorrections
pubmed:language
eng
pubmed:journal
pubmed:status
PubMed-not-MEDLINE
pubmed:month
Aug
pubmed:issn
0022-1007
pubmed:author
pubmed:issnType
Print
pubmed:day
31
pubmed:volume
74
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
167-75
pubmed:dateRevised
2010-9-28
pubmed:year
1941
pubmed:articleTitle
THE CAPACITY OF THE RENAL VASCULAR BED IN HYPERTENSION.
pubmed:affiliation
Department of Pathology, Stanford University School of Medicine, San Francisco.
pubmed:publicationType
Journal Article