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| pubmed-article:19855253 | lifeskim:mentions | umls-concept:C0237401 | lld:lifeskim |
| pubmed-article:19855253 | lifeskim:mentions | umls-concept:C0599755 | lld:lifeskim |
| pubmed-article:19855253 | lifeskim:mentions | umls-concept:C0376705 | lld:lifeskim |
| pubmed-article:19855253 | lifeskim:mentions | umls-concept:C0963057 | lld:lifeskim |
| pubmed-article:19855253 | lifeskim:mentions | umls-concept:C1411976 | lld:lifeskim |
| pubmed-article:19855253 | lifeskim:mentions | umls-concept:C1882417 | lld:lifeskim |
| pubmed-article:19855253 | pubmed:issue | 18 | lld:pubmed |
| pubmed-article:19855253 | pubmed:dateCreated | 2009-11-12 | lld:pubmed |
| pubmed-article:19855253 | pubmed:abstractText | Toll-like receptors (TLRs) are innate immune sensors that are integral to resisting chronic and opportunistic infections. Mounting evidence implicates TLR polymorphisms in susceptibilities to various infectious diseases, including HIV-1. We investigated the impact of TLR single nucleotide polymorphisms (SNPs) on clinical outcome in a seroincident cohort of HIV-1-infected volunteers. | lld:pubmed |
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| pubmed-article:19855253 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:19855253 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:19855253 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:19855253 | pubmed:month | Nov | lld:pubmed |
| pubmed-article:19855253 | pubmed:issn | 1473-5571 | lld:pubmed |
| pubmed-article:19855253 | pubmed:author | pubmed-author:McElrathM... | lld:pubmed |
| pubmed-article:19855253 | pubmed:author | pubmed-author:BochudPierre-... | lld:pubmed |
| pubmed-article:19855253 | pubmed:author | pubmed-author:PineSamuel... | lld:pubmed |
| pubmed-article:19855253 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:19855253 | pubmed:day | 27 | lld:pubmed |
| pubmed-article:19855253 | pubmed:volume | 23 | lld:pubmed |
| pubmed-article:19855253 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:19855253 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:19855253 | pubmed:pagination | 2387-95 | lld:pubmed |
| pubmed-article:19855253 | pubmed:dateRevised | 2011-2-14 | lld:pubmed |
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| pubmed-article:19855253 | pubmed:year | 2009 | lld:pubmed |
| pubmed-article:19855253 | pubmed:articleTitle | Polymorphisms in toll-like receptor 4 and toll-like receptor 9 influence viral load in a seroincident cohort of HIV-1-infected individuals. | lld:pubmed |
| pubmed-article:19855253 | pubmed:affiliation | Program in Pathobiology, Department of Global Health, University of Washington, Seattle, Washington, USA. | lld:pubmed |
| pubmed-article:19855253 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:19855253 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| pubmed-article:19855253 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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