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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
1
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pubmed:dateCreated |
2010-1-21
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pubmed:abstractText |
Excess accumulation of vascular extracellular matrix (ECM) is an important pathological process in cardiovascular diseases including diabetes-associated atherosclerosis. We explored how a recently identified molecule, cell division autoantigen 1 (CDA1), influences the profibrotic TGF-beta pathway leading to vascular ECM accumulation.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
http://linkedlifedata.com/resource/pubmed/chemical/Autoantigens,
http://linkedlifedata.com/resource/pubmed/chemical/Blood Glucose,
http://linkedlifedata.com/resource/pubmed/chemical/Connective Tissue Growth Factor,
http://linkedlifedata.com/resource/pubmed/chemical/Ctgf protein, mouse,
http://linkedlifedata.com/resource/pubmed/chemical/Hemoglobin A, Glycosylated,
http://linkedlifedata.com/resource/pubmed/chemical/Lipoproteins,
http://linkedlifedata.com/resource/pubmed/chemical/Luciferases,
http://linkedlifedata.com/resource/pubmed/chemical/RNA, Messenger,
http://linkedlifedata.com/resource/pubmed/chemical/RNA, Small Interfering,
http://linkedlifedata.com/resource/pubmed/chemical/Transforming Growth Factor beta,
http://linkedlifedata.com/resource/pubmed/chemical/cell division autoantigen-1
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pubmed:status |
MEDLINE
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pubmed:month |
Jan
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pubmed:issn |
1432-0428
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pubmed:author |
pubmed-author:AllenT JTJ,
pubmed-author:CalkinA CAC,
pubmed-author:CaoZZ,
pubmed-author:ChanEE,
pubmed-author:CooperM EME,
pubmed-author:Jandeleit-DahmK AKA,
pubmed-author:OOIS KSK,
pubmed-author:PhamYY,
pubmed-author:TohB-HBH,
pubmed-author:TuYY,
pubmed-author:WatsonA MAM,
pubmed-author:WuTT
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pubmed:issnType |
Electronic
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pubmed:volume |
53
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
170-9
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pubmed:meshHeading |
pubmed-meshheading:19847393-Animals,
pubmed-meshheading:19847393-Aorta,
pubmed-meshheading:19847393-Atherosclerosis,
pubmed-meshheading:19847393-Autoantigens,
pubmed-meshheading:19847393-Blood Glucose,
pubmed-meshheading:19847393-Connective Tissue Growth Factor,
pubmed-meshheading:19847393-Diabetes Mellitus, Experimental,
pubmed-meshheading:19847393-Diabetic Angiopathies,
pubmed-meshheading:19847393-Extracellular Matrix,
pubmed-meshheading:19847393-Gene Expression Regulation,
pubmed-meshheading:19847393-Genes, Reporter,
pubmed-meshheading:19847393-Hemoglobin A, Glycosylated,
pubmed-meshheading:19847393-Lipoproteins,
pubmed-meshheading:19847393-Luciferases,
pubmed-meshheading:19847393-Mice,
pubmed-meshheading:19847393-RNA, Messenger,
pubmed-meshheading:19847393-RNA, Small Interfering,
pubmed-meshheading:19847393-Reverse Transcriptase Polymerase Chain Reaction,
pubmed-meshheading:19847393-Signal Transduction,
pubmed-meshheading:19847393-Transforming Growth Factor beta
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pubmed:year |
2010
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pubmed:articleTitle |
Cell division autoantigen 1 plays a profibrotic role by modulating downstream signalling of TGF-beta in a murine diabetic model of atherosclerosis.
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pubmed:affiliation |
Diabetes and Metabolism Division, Baker IDI Heart and Diabetes Institute, Melbourne, Victoria 3004, Australia.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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