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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
1
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pubmed:dateCreated |
2010-1-21
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pubmed:abstractText |
Helicobacter pylori (HP) infection potently induces aberrant DNA methylation in gastric mucosae, and its accumulation is associated with gastric cancer risk. Cross-sectional analysis of methylation levels (fraction of methylated DNA molecules) and temporal analysis of methylation incidence suggested that methylation levels decrease after HP infection discontinues. We aimed to demonstrate the decrease in methylation levels.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:status |
MEDLINE
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pubmed:issn |
1435-5922
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pubmed:author |
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pubmed:issnType |
Electronic
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pubmed:volume |
45
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
37-44
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pubmed:meshHeading |
pubmed-meshheading:19821005-Aged,
pubmed-meshheading:19821005-Case-Control Studies,
pubmed-meshheading:19821005-Cross-Sectional Studies,
pubmed-meshheading:19821005-DNA Methylation,
pubmed-meshheading:19821005-Female,
pubmed-meshheading:19821005-Gastric Mucosa,
pubmed-meshheading:19821005-Helicobacter Infections,
pubmed-meshheading:19821005-Helicobacter pylori,
pubmed-meshheading:19821005-Humans,
pubmed-meshheading:19821005-Inflammation,
pubmed-meshheading:19821005-Male,
pubmed-meshheading:19821005-Middle Aged,
pubmed-meshheading:19821005-Polymerase Chain Reaction,
pubmed-meshheading:19821005-Stomach Neoplasms,
pubmed-meshheading:19821005-Time Factors
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pubmed:year |
2010
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pubmed:articleTitle |
Persistence of a component of DNA methylation in gastric mucosae after Helicobacter pylori eradication.
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pubmed:affiliation |
Carcinogenesis Division, National Cancer Center Research Institute, 5-1-1 Tsukiji, Chuo-ku, Tokyo 104-0045, Japan. tnakajim@ncc.go.jp
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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