| pubmed-article:19812523 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:19812523 | lifeskim:mentions | umls-concept:C0346647 | lld:lifeskim |
| pubmed-article:19812523 | lifeskim:mentions | umls-concept:C0596244 | lld:lifeskim |
| pubmed-article:19812523 | lifeskim:mentions | umls-concept:C1412236 | lld:lifeskim |
| pubmed-article:19812523 | lifeskim:mentions | umls-concept:C1412237 | lld:lifeskim |
| pubmed-article:19812523 | lifeskim:mentions | umls-concept:C1413867 | lld:lifeskim |
| pubmed-article:19812523 | lifeskim:mentions | umls-concept:C0678951 | lld:lifeskim |
| pubmed-article:19812523 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:19812523 | pubmed:dateCreated | 2010-2-25 | lld:pubmed |
| pubmed-article:19812523 | pubmed:abstractText | Pancreatic carcinoma etiology and molecular pathogenesis are weakly understood. Based on the assumption that genetic variation in carcinogen metabolism further modifies the risk of exposure-related cancers, we studied the association of polymorphisms in the tobacco carcinogen-metabolizing gene CYP2A13 (Arg101Stop) and the alcohol-metabolizing genes ADH1B (Arg48His) and ADH1C (Ile350Val) with pancreatic cancer risk. | lld:pubmed |
| pubmed-article:19812523 | pubmed:language | eng | lld:pubmed |
| pubmed-article:19812523 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19812523 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:19812523 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:19812523 | pubmed:month | Mar | lld:pubmed |
| pubmed-article:19812523 | pubmed:issn | 1536-4828 | lld:pubmed |
| pubmed-article:19812523 | pubmed:author | pubmed-author:SoucekPavelP | lld:pubmed |
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| pubmed-article:19812523 | pubmed:author | pubmed-author:VranaDavidD | lld:pubmed |
| pubmed-article:19812523 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:19812523 | pubmed:volume | 39 | lld:pubmed |
| pubmed-article:19812523 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:19812523 | pubmed:authorsComplete | Y | lld:pubmed |
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| pubmed-article:19812523 | pubmed:year | 2010 | lld:pubmed |
| pubmed-article:19812523 | pubmed:articleTitle | CYP2A13, ADH1B, and ADH1C gene polymorphisms and pancreatic cancer risk. | lld:pubmed |
| pubmed-article:19812523 | pubmed:affiliation | Toxicogenomics Unit, National Institute of Public Health, Prague, Czech Republic. | lld:pubmed |
| pubmed-article:19812523 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:19812523 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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