| pubmed-article:19745198 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:19745198 | lifeskim:mentions | umls-concept:C0030190 | lld:lifeskim |
| pubmed-article:19745198 | lifeskim:mentions | umls-concept:C0151942 | lld:lifeskim |
| pubmed-article:19745198 | lifeskim:mentions | umls-concept:C0013081 | lld:lifeskim |
| pubmed-article:19745198 | lifeskim:mentions | umls-concept:C0547070 | lld:lifeskim |
| pubmed-article:19745198 | lifeskim:mentions | umls-concept:C1947917 | lld:lifeskim |
| pubmed-article:19745198 | lifeskim:mentions | umls-concept:C0392756 | lld:lifeskim |
| pubmed-article:19745198 | lifeskim:mentions | umls-concept:C0599946 | lld:lifeskim |
| pubmed-article:19745198 | lifeskim:mentions | umls-concept:C0291751 | lld:lifeskim |
| pubmed-article:19745198 | pubmed:issue | 12 | lld:pubmed |
| pubmed-article:19745198 | pubmed:dateCreated | 2009-11-20 | lld:pubmed |
| pubmed-article:19745198 | pubmed:abstractText | Reduced fibrinolytic activity is associated with adverse cardiovascular events. Although insulin-regulated aminopeptidase (IRAP) was recently identified as the angiotensin (Ang) IV receptor (AT4R), the impact of AngIV-AT4R signaling distal to AngII on the activation of type-1 plasminogen activator inhibitor (PAI-1) in the fibrinolytic process and subsequent formation of thrombosis remains unclarified. | lld:pubmed |
| pubmed-article:19745198 | pubmed:language | eng | lld:pubmed |
| pubmed-article:19745198 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19745198 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:19745198 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:19745198 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:19745198 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:19745198 | pubmed:month | Dec | lld:pubmed |
| pubmed-article:19745198 | pubmed:issn | 1524-4636 | lld:pubmed |
| pubmed-article:19745198 | pubmed:author | pubmed-author:YamamotoKojiK | lld:pubmed |
| pubmed-article:19745198 | pubmed:author | pubmed-author:IshiiMasakazu... | lld:pubmed |
| pubmed-article:19745198 | pubmed:author | pubmed-author:NumaguchiYasu... | lld:pubmed |
| pubmed-article:19745198 | pubmed:author | pubmed-author:OkumuraKenjiK | lld:pubmed |
| pubmed-article:19745198 | pubmed:author | pubmed-author:KubotaRyujiR | lld:pubmed |
| pubmed-article:19745198 | pubmed:author | pubmed-author:MuroharaToyoa... | lld:pubmed |
| pubmed-article:19745198 | pubmed:author | pubmed-author:MatsushitaTad... | lld:pubmed |
| pubmed-article:19745198 | pubmed:author | pubmed-author:MoritaYasuhir... | lld:pubmed |
| pubmed-article:19745198 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:19745198 | pubmed:volume | 29 | lld:pubmed |
| pubmed-article:19745198 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:19745198 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:19745198 | pubmed:pagination | 2102-8 | lld:pubmed |
| pubmed-article:19745198 | pubmed:dateRevised | 2010-11-18 | lld:pubmed |
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| pubmed-article:19745198 | pubmed:meshHeading | pubmed-meshheading:19745198... | lld:pubmed |
| pubmed-article:19745198 | pubmed:year | 2009 | lld:pubmed |
| pubmed-article:19745198 | pubmed:articleTitle | Ablation of angiotensin IV receptor attenuates hypofibrinolysis via PAI-1 downregulation and reduces occlusive arterial thrombosis. | lld:pubmed |
| pubmed-article:19745198 | pubmed:affiliation | Department of Medical Science of Proteases, Nagoya University School of Medicine, 65 Tsurumai, Showa-ku, Nagoya 466-8550, Japan. numa2@med.nagoya-u.ac.jp | lld:pubmed |
| pubmed-article:19745198 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:19745198 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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