Linked Life Data

19648062

Source:http://linkedlifedata.com/resource/pubmed/id/19648062

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
9
pubmed:dateCreated
2009-8-31
pubmed:abstractText
Mutations in the SCN5A gene, which encodes the cardiac sodium channel, have been implicated in the pathogenesis of Brugada syndrome (BrS). Febrile illnesses have been recognized to unmask and/or trigger the BrS phenotype. However, the pathophysiological mechanism has not been fully elucidated.
pubmed:grant
pubmed:commentsCorrections
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pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Sep
pubmed:issn
1556-3871
pubmed:author
pubmed:issnType
Electronic
pubmed:volume
6
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
1318-26
pubmed:dateRevised
2011-7-22
pubmed:meshHeading
pubmed:year
2009
pubmed:articleTitle
A novel SCN5A mutation V1340I in Brugada syndrome augmenting arrhythmias during febrile illness.
pubmed:affiliation
Electrophysiology Research Laboratory, Texas Heart Institute/St. Luke's Episcopal Hospital, Houston, Texas 77030, USA.
pubmed:publicationType
Journal Article, Case Reports