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rdf:type |
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lifeskim:mentions |
umls-concept:C0015688,
umls-concept:C0020538,
umls-concept:C0051696,
umls-concept:C0087111,
umls-concept:C0681850,
umls-concept:C0948265,
umls-concept:C1327413,
umls-concept:C1550501,
umls-concept:C1706203,
umls-concept:C2349001,
umls-concept:C2697811
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pubmed:issue |
4
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pubmed:dateCreated |
2009-8-21
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pubmed:abstractText |
Angiotensin II increases activation of oxidative signaling and vascular inflammatory gene expression, and interruption of the renin-angiotensin system has been considered more vasculoprotective than use of calcium channel antagonists and other anti-hypertensive therapies. Despite these putative mechanisms, amlodipine is equally efficacious as other therapies in reducing cardiovascular events.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
http://linkedlifedata.com/resource/pubmed/chemical/Amlodipine,
http://linkedlifedata.com/resource/pubmed/chemical/Angiotensin II Type 1 Receptor...,
http://linkedlifedata.com/resource/pubmed/chemical/Antihypertensive Agents,
http://linkedlifedata.com/resource/pubmed/chemical/Cell Adhesion Molecules,
http://linkedlifedata.com/resource/pubmed/chemical/Cytokines,
http://linkedlifedata.com/resource/pubmed/chemical/Fatty Acids, Nonesterified,
http://linkedlifedata.com/resource/pubmed/chemical/Imidazoles,
http://linkedlifedata.com/resource/pubmed/chemical/Lipopolysaccharides,
http://linkedlifedata.com/resource/pubmed/chemical/Tetrazoles,
http://linkedlifedata.com/resource/pubmed/chemical/olmesartan medoxomil
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pubmed:status |
MEDLINE
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pubmed:month |
Aug
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pubmed:issn |
1573-7241
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pubmed:author |
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pubmed:issnType |
Electronic
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pubmed:volume |
23
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
289-94
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pubmed:meshHeading |
pubmed-meshheading:19641984-Adult,
pubmed-meshheading:19641984-Aged,
pubmed-meshheading:19641984-Amlodipine,
pubmed-meshheading:19641984-Angiotensin II Type 1 Receptor Blockers,
pubmed-meshheading:19641984-Antihypertensive Agents,
pubmed-meshheading:19641984-Cell Adhesion Molecules,
pubmed-meshheading:19641984-Cytokines,
pubmed-meshheading:19641984-Double-Blind Method,
pubmed-meshheading:19641984-Fatty Acids, Nonesterified,
pubmed-meshheading:19641984-Female,
pubmed-meshheading:19641984-Humans,
pubmed-meshheading:19641984-Hypertension,
pubmed-meshheading:19641984-Imidazoles,
pubmed-meshheading:19641984-Lipopolysaccharides,
pubmed-meshheading:19641984-Male,
pubmed-meshheading:19641984-Metabolic Syndrome X,
pubmed-meshheading:19641984-Middle Aged,
pubmed-meshheading:19641984-Oxidation-Reduction,
pubmed-meshheading:19641984-Oxidative Stress,
pubmed-meshheading:19641984-Tetrazoles
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pubmed:year |
2009
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pubmed:articleTitle |
Treatment of hypertension in metabolic syndrome subjects with amlodipine and olmesartan-effects on oxidized non-esterified free fatty acids and cytokine production.
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pubmed:affiliation |
Department of Medicine, SUNY Downstate, Brooklyn, NY 11203, USA. robert.rosenson@downstate.edu
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pubmed:publicationType |
Journal Article,
Comparative Study,
Randomized Controlled Trial,
Research Support, Non-U.S. Gov't
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