19604877

Source:http://linkedlifedata.com/resource/pubmed/id/19604877

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Predicate	Object
rdf:type	pubmed:Citation
lifeskim:mentions	umls-concept:C0013227, umls-concept:C0034693, umls-concept:C0034721, umls-concept:C0430797, umls-concept:C0680242
pubmed:issue	3
pubmed:dateCreated	2009-7-16
pubmed:abstractText	Administration of gamma-hydroxybutyrate (GHB) (200 mg/kg i.p.) in rats evoked a hypersynchronous electrocorticogram pattern which was antagonized by specific anti-petit mal agents, while other antiepileptic drugs exerted no influence, or even potentiated the effect of GHB. The specific sensitivity of the GHB-induced hypersynchrony to anti-petit mal agents suggests a possible use of this effect as a model for testing potential anti-petit mal agents.
pubmed:language	eng
pubmed:journal	http://linkedlifedata.com/resource/pubmed/journal/7600130
pubmed:status	PubMed-not-MEDLINE
pubmed:month	Oct
pubmed:issn	0304-3940
pubmed:author	pubmed-author:BontaI LIL, pubmed-author:DzoljicM RMR, pubmed-author:GodschalkMM
pubmed:issnType	Print
pubmed:volume	3
pubmed:owner	NLM
pubmed:authorsComplete	Y
pubmed:pagination	145-50
pubmed:year	1976
pubmed:articleTitle	Antagonism of gamma-hydroxybutyrate-induced hypersynchronization in the ECoG of the rat by anti-petit mal drugs.
pubmed:affiliation	Department of Pharmacology, Faculty of Medicine, Erasmus University Rotterdam, P.O. Box 1738, Rotterdam, The Netherlands.
pubmed:publicationType	Journal Article